NFATc3 is required for intermittent hypoxia-induced hypertension. - Wikidata - awgldk - TriplyDB

NFATc3 is required for intermittent hypoxia-induced hypertension.

NFATc3 is required for intermittent hypoxia-induced hypertension.

http://www.wikidata.org/entity/Q42410702

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Mechanisms of diesel-induced endothelial nitric oxide synthase dysfunction in coronary arterioles Preproendothelin-1 expression is negatively regulated by IFNγ during hepatic stellate cell activation Pathophysiology of sleep apnea NFATc3 contributes to intermittent hypoxia-induced arterial remodeling in mice Endothelin-1 contributes to increased NFATc3 activation by chronic hypoxia in pulmonary arteries.NFATc3 is required for chronic hypoxia-induced pulmonary hypertension in adult and neonatal mice Resveratrol reverses monocrotaline-induced pulmonary vascular and cardiac dysfunction: a potential role for atrogin-1 in smooth muscle Transcriptional responses to intermittent hypoxia NFATc3 and VIP in Idiopathic Pulmonary Fibrosis and Chronic Obstructive Pulmonary Disease NFAT is required for spontaneous pulmonary hypertension in superoxide dismutase 1 knockout mice.ASIC1-mediated calcium entry stimulates NFATc3 nuclear translocation via PICK1 coupling in pulmonary arterial smooth muscle cells Parsing good versus bad signaling pathways in the heart: role of calcineurin-nuclear factor of activated T-cells.Intermittent hypoxia-induced increases in reactive oxygen species activate NFATc3 increasing endothelin-1 vasoconstrictor reactivity.Mechanisms of intermittent hypoxia induced hypertension.Endothelin-1-induced vasoconstriction does not require intracellular Ca²⁺ waves in arteries from rats exposed to intermittent hypoxia.Aquaporin-2 promoter is synergistically regulated by nitric oxide and nuclear factor of activated T cells.Heparanase Inhibition Reduces Glucose Levels, Blood Pressure, and Oxidative Stress in Apolipoprotein E Knockout Mice.NFAT regulation of cystathionine γ-lyase expression in endothelial cells is impaired in rats exposed to intermittent hypoxia.Effect of chronic continual- and intermittent hypoxia-induced systemic inflammation on the cardiovascular system in rats.Cardiovascular consequences of sleep apnea.PLCγ1-PKCε-IP3R1 plays an important role in hypoxia-induced calcium response in pulmonary artery smooth muscle cells.Fasudil improves endothelial dysfunction in rats exposed to chronic intermittent hypoxia through RhoA/ROCK/NFATc3 pathway.

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NFATc3 is required for intermittent hypoxia-induced hypertension.

http://www.wikidata.org/entity/Q42410702

description

2008 nî lūn-bûn

@nan

2008年の論文

@ja

2008年論文

@yue

2008年論文

@zh-hant

2008年論文

@zh-hk

2008年論文

@zh-mo

2008年論文

@zh-tw

2008年论文

@wuu

2008年论文

@zh

2008年论文

@zh-cn

name

NFATc3 is required for intermittent hypoxia-induced hypertension.

@en

NFATc3 is required for intermittent hypoxia-induced hypertension.

@nl

type

label

NFATc3 is required for intermittent hypoxia-induced hypertension.

@en

NFATc3 is required for intermittent hypoxia-induced hypertension.

@nl

prefLabel

NFATc3 is required for intermittent hypoxia-induced hypertension.

@en

NFATc3 is required for intermittent hypoxia-induced hypertension.

@nl

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AJPHEART.00132.2008

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American Journal of Physiology - Heart and Circulatory Physiology

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NFATc3 is required for intermittent hypoxia-induced hypertension.

@en

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Dominique Alò

http://www.w3.org/2001/XMLSchema#string

Laura Duling

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Laura González Bosc

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Mary Walker

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Nancy Kanagy

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Sergio de Frutos

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Tammy Berry

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P2860

Calcineurin-GATA-6 pathway is involved in smooth muscle-specific transcription.

Targeted disruption of NFATc3, but not NFATc4, reveals an intrinsic defect in calcineurin-mediated cardiac hypertrophic growth

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Signals transduced by Ca(2+)/calcineurin and NFATc3/c4 pattern the developing vasculature

Transcription factors of the NFAT family: regulation and function

NFATc3 mediates chronic hypoxia-induced pulmonary arterial remodeling with alpha-actin up-regulation

The nuclear factor of activated T cells in pulmonary arterial hypertension can be therapeutically targeted

Estradiol attenuates hypoxia-induced pulmonary endothelin-1 gene expression

Augmented endothelin vasoconstriction in intermittent hypoxia-induced hypertension

Differential effects of chronic hypoxia and intermittent hypocapnic and eucapnic hypoxia on pulmonary vasoreactivity

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H2382-90

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scholarly article

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10.1152/AJPHEART.00132.2008

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5

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294

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Olan Jackson-Weaver

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2008-03-21T00:00:00Z

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5492292

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2953847

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