Dominant negative mutants of the murine cytomegalovirus M53 gene block nuclear egress and inhibit capsid maturation.
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The Herpes Simplex Virus Protein pUL31 Escorts Nucleocapsids to Sites of Nuclear Egress, a Process Coordinated by Its N-Terminal Domain.Cytomegalovirus replicon-based regulation of gene expression in vitro and in vivo.M94 is essential for the secondary envelopment of murine cytomegalovirus.Real-time transcriptional profiling of cellular and viral gene expression during lytic cytomegalovirus infectionA modified screening system for loss-of-function and dominant negative alleles of essential MCMV genes.A cytomegalovirus-based vaccine expressing a single tumor-specific CD8+ T-cell epitope delays tumor growth in a murine model of prostate cancerUnexpected features and mechanism of heterodimer formation of a herpesvirus nuclear egress complexInteractions of the Kaposi's Sarcoma-associated herpesvirus nuclear egress complex: ORF69 is a potent factor for remodeling cellular membranesThe human cytomegalovirus nuclear egress complex unites multiple functions: Recruitment of effectors, nuclear envelope rearrangement, and docking to nuclear capsids.Functional characterization of nuclear trafficking signals in pseudorabies virus pUL31.Intragenic and extragenic suppression of a mutation in herpes simplex virus 1 UL34 that affects both nuclear envelope targeting and membrane budding.A physical link between the pseudorabies virus capsid and the nuclear egress complex.Identification of conserved amino acids in pUL34 which are critical for function of the pseudorabies virus nuclear egress complex.Human cytomegalovirus UL50 and UL53 recruit viral protein kinase UL97, not protein kinase C, for disruption of nuclear lamina and nuclear egress in infected cellsGetting to and through the inner nuclear membrane during herpesvirus nuclear egress.Characterization of conserved region 2-deficient mutants of the cytomegalovirus egress protein pM53.Nuclear egress of pseudorabies virus capsids is enhanced by a subspecies of the large tegument protein that is lost upon cytoplasmic maturationStructural determinants for nuclear envelope localization and function of pseudorabies virus pUL34.Herpes simplex virus 1 pUL34 plays a critical role in cell-to-cell spread of virus in addition to its role in virus replicationThe Herpesvirus Nuclear Egress Complex Component, UL31, Can Be Recruited to Sites of DNA Damage Through Poly-ADP Ribose Binding.Human cytomegalovirus UL97 phosphorylates the viral nuclear egress complexVenture from the Interior-Herpesvirus pUL31 Escorts Capsids from Nucleoplasmic Replication Compartments to Sites of Primary Envelopment at the Inner Nuclear Membrane.Mouse cytomegalovirus egress protein pM50 interacts with cellular endophilin-A2
P2860
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P2860
Dominant negative mutants of the murine cytomegalovirus M53 gene block nuclear egress and inhibit capsid maturation.
description
2010 nî lūn-bûn
@nan
2010年の論文
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2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
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2010年论文
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2010年论文
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name
Dominant negative mutants of t ...... and inhibit capsid maturation.
@en
Dominant negative mutants of t ...... and inhibit capsid maturation.
@nl
type
label
Dominant negative mutants of t ...... and inhibit capsid maturation.
@en
Dominant negative mutants of t ...... and inhibit capsid maturation.
@nl
prefLabel
Dominant negative mutants of t ...... and inhibit capsid maturation.
@en
Dominant negative mutants of t ...... and inhibit capsid maturation.
@nl
P2093
P2860
P356
P1433
P1476
Dominant negative mutants of t ...... and inhibit capsid maturation
@en
P2093
Lars Dölken
Mark Lötzerich
Mirela Popa
Paul Walther
Ulrich H Koszinowski
Zsolt Ruzsics
P2860
P304
P356
10.1128/JVI.00681-10
P407
P577
2010-07-07T00:00:00Z