Persistent hyperalgesia in the cisplatin-treated mouse as defined by threshold measures, the conditioned place preference paradigm, and changes in dorsal root ganglia activated transcription factor 3: the effects of gabapentin, ketorolac, and etaner
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The search for novel analgesics: targets and mechanismsMetformin Prevents Cisplatin-Induced Cognitive Impairment and Brain Damage in MiceWelfare Assessment following Heterotopic or Orthotopic Inoculation of Bladder Cancer in C57BL/6 MicePathophysiology of Chemotherapy-Induced Peripheral Neuropathy.The conditioned place preference test for assessing welfare consequences and potential refinements in a mouse bladder cancer model.Soluble epoxide hydrolase inhibition is antinociceptive in a mouse model of diabetic neuropathy.Reward and motivation in pain and pain reliefGabapentin alleviates affective pain after traumatic nerve injury.A New Rat Model of Cisplatin-induced Neuropathic PainDifferences in cisplatin-induced mechanical allodynia in male and female mice.Chemotherapy-induced painful neuropathy: pain-like behaviours in rodent models and their response to commonly used analgesicsToll-like receptor signaling regulates cisplatin-induced mechanical allodynia in miceChemotherapy induced peripheral neuropathic painInflammatory 'double hit' model of temporomandibular joint disorder with elevated CCL2, CXCL9, CXCL10, RANTES and behavioural hypersensitivity in TNFR1/R2-/- mice.Multiple sites and actions of gabapentin-induced relief of ongoing experimental neuropathic pain.Analgesic effects of clinically used compounds in novel mouse models of polyneuropathy induced by oxaliplatin and cisplatin.Cisplatin induced sensory neuropathy is prevented by vascular endothelial growth factor-A.The effect of gabapentin and ketorolac on allodynia and conditioned place preference in antibody-induced inflammation.Cancer Chemotherapy in Early Life Significantly Alters the Maturation of Pain Processing.Blocking of cytokines signalling attenuates evoked and spontaneous neuropathic pain behaviours in the paclitaxel rat model of chemotherapy-induced neuropathy.Identification of FAM173B as a protein methyltransferase promoting chronic pain.HDAC6 inhibition effectively reverses chemotherapy-induced peripheral neuropathy.Monoacylglycerol lipase inhibitors reverse paclitaxel-induced nociceptive behavior and proinflammatory markers in a mouse model of chemotherapy-induced neuropathy.
P2860
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P2860
Persistent hyperalgesia in the cisplatin-treated mouse as defined by threshold measures, the conditioned place preference paradigm, and changes in dorsal root ganglia activated transcription factor 3: the effects of gabapentin, ketorolac, and etaner
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Persistent hyperalgesia in the ...... apentin, ketorolac, and etaner
@en
Persistent hyperalgesia in the ...... apentin, ketorolac, and etaner
@nl
type
label
Persistent hyperalgesia in the ...... apentin, ketorolac, and etaner
@en
Persistent hyperalgesia in the ...... apentin, ketorolac, and etaner
@nl
prefLabel
Persistent hyperalgesia in the ...... apentin, ketorolac, and etaner
@en
Persistent hyperalgesia in the ...... apentin, ketorolac, and etaner
@nl
P2093
P2860
P1476
Persistent hyperalgesia in the ...... apentin, ketorolac, and etaner
@en
P2093
Elaine Pirie
Hue Jung Park
James Skahen
Jennifer A Stokes
Tony L Yaksh
Yuri Shtaerman
P2860
P304
P356
10.1213/ANE.0B013E31826E1007
P407
P577
2012-12-07T00:00:00Z