Constitutive activation of RANK disrupts mammary cell fate leading to tumorigenesis. - Wikidata - awgldk - TriplyDB

Constitutive activation of RANK disrupts mammary cell fate leading to tumorigenesis.

Constitutive activation of RANK disrupts mammary cell fate leading to tumorigenesis.

http://www.wikidata.org/entity/Q42439949

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Targeting RANKL in metastasis Mammary gland stem cells and their application in breast cancer BRCA1 haploinsufficiency cell-autonomously activates RANKL expression and generates denosumab-responsive breast cancer-initiating cells.Aberrant Activation of the RANK Signaling Receptor Induces Murine Salivary Gland Tumors.Aberrant activation of NF-κB signaling in mammary epithelium leads to abnormal growth and ductal carcinoma in situ.The seventh ENBDC workshop on methods in mammary gland development and cancer RANK Signaling Amplifies WNT-Responsive Mammary Progenitors through R-SPONDIN1.Breast cancer stem cells and the immune system: promotion, evasion and therapy.Hormone-sensing mammary epithelial progenitors: emerging identity and hormonal regulation.RANK as a therapeutic target in cancer.Receptor activator of nuclear factor kappa B (RANK) expression in primary breast cancer correlates with recurrence-free survival and development of bone metastases in I-SPY1 (CALGB 150007/150012; ACRIN 6657).Cancer stem cell signaling pathways.Cell lineage determinants as regulators of breast cancer metastasis.Rankl Impairs Lactogenic Differentiation Through Inhibition of the Prolactin/Stat5 Pathway at Midgestation.Key Factors in Breast Cancer Dissemination and Establishment at the Bone: Past, Present and Future Perspectives.Mammary Stem Cells and Breast Cancer Stem Cells: Molecular Connections and Clinical Implications

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P2860

Constitutive activation of RANK disrupts mammary cell fate leading to tumorigenesis.

http://www.wikidata.org/entity/Q42439949

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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2013年论文

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2013年论文

@zh-cn

name

Constitutive activation of RANK disrupts mammary cell fate leading to tumorigenesis.

@en

Constitutive activation of RANK disrupts mammary cell fate leading to tumorigenesis.

@nl

type

label

Constitutive activation of RANK disrupts mammary cell fate leading to tumorigenesis.

@en

Constitutive activation of RANK disrupts mammary cell fate leading to tumorigenesis.

@nl

prefLabel

Constitutive activation of RANK disrupts mammary cell fate leading to tumorigenesis.

@en

Constitutive activation of RANK disrupts mammary cell fate leading to tumorigenesis.

@nl

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Constitutive activation of RANK disrupts mammary cell fate leading to tumorigenesis

@en

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Alex Cordero

http://www.w3.org/2001/XMLSchema#string

Marta Ines Gallego

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Miguel Angel Pujana

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Pasquale Pellegrini

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Purificación Muñoz

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William C Dougall

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P2860

Comprehensive molecular portraits of human breast tumours

Systematic and integrative analysis of large gene lists using DAVID bioinformatics resources

The osteoclast differentiation factor osteoprotegerin-ligand is essential for mammary gland development

RANK ligand mediates progestin-induced mammary epithelial proliferation and carcinogenesis

Expression of the neu protooncogene in the mammary epithelium of transgenic mice induces metastatic disease.

PML depletion disrupts normal mammary gland development and skews the composition of the mammary luminal cell progenitor pool

Elf5 inhibits the epithelial–mesenchymal transition in mammary gland development and breast cancer metastasis by transcriptionally repressing Snail2

Osteoclast differentiation and activation

Generation of a functional mammary gland from a single stem cell

Supervised risk predictor of breast cancer based on intrinsic subtypes

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1954-1965

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10.1002/STEM.1454

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9

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31

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Eva Gonzalez-Suarez

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2013-09-01T00:00:00Z

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237844425

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