Interaction of PKC and NOS in signal transduction of microvascular hyperpermeability.
about
VE-cadherin and beta-catenin binding dynamics during histamine-induced endothelial hyperpermeabilityA vascular gene trap screen defines RasGRP3 as an angiogenesis-regulated gene required for the endothelial response to phorbol estersInvolvement of the H1 Histamine Receptor, p38 MAP Kinase, Myosin Light Chains Kinase, and Rho/ROCK in Histamine-Induced Endothelial Barrier Dysfunction.Moderate exercise promotes human RBC-NOS activity, NO production and deformability through Akt kinase pathway.Dyslipidemia and diabetic retinopathy.TRPC-mediated actin-myosin contraction is critical for BBB disruption following hypoxic stress.The NO cascade, eNOS location, and microvascular permeability.The Ras activator RasGRP3 mediates diabetes-induced embryonic defects and affects endothelial cell migration.ROCK and cAMP promote lymphatic endothelial cell barrier integrity and modulate histamine and thrombin-induced barrier dysfunction.Harvesting, identification and barrier function of human lung microvascular endothelial cellsProtein kinase C inhibition and diabetic retinopathy: a shot in the dark at translational researchBixa orellana leaf extract suppresses histamine-induced endothelial hyperpermeability via the PLC-NO-cGMP signaling cascade.PKCδ impaired vessel formation and angiogenic factor expression in diabetic ischemic limbs.Myosin light chain kinase signaling in endothelial barrier dysfunction.Cellular signaling and potential new treatment targets in diabetic retinopathy.Inhibition of neuronal nitric oxide synthase in ovine model of acute lung injury.Nitric oxide, S-nitrosation, and endothelial permeability.Regulation of pulmonary endothelial barrier function by kinases.HIV-1 gp120 compromises blood-brain barrier integrity and enhances monocyte migration across blood-brain barrier: implication for viral neuropathogenesis.Diabetic retinopathy: Breaking the barrier.IGFBP-3 reduces eNOS and PKCzeta phosphorylation, leading to lowered VEGF levels.The protein kinase MEK1/2 mediate vascular endothelial growth factor- and histamine-induced hyperpermeability in porcine coronary venules.Protein Kinase C as Regulator of Vascular Smooth Muscle Function and Potential Target in Vascular Disorders.Evolving Mechanisms of Vascular Smooth Muscle Contraction Highlight Key Targets in Vascular Disease.Flow modulates the transport of K+ through the walls of single perfused mesenteric venules in anaesthetised rats.
P2860
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P2860
Interaction of PKC and NOS in signal transduction of microvascular hyperpermeability.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Interaction of PKC and NOS in signal transduction of microvascular hyperpermeability.
@en
Interaction of PKC and NOS in signal transduction of microvascular hyperpermeability.
@nl
type
label
Interaction of PKC and NOS in signal transduction of microvascular hyperpermeability.
@en
Interaction of PKC and NOS in signal transduction of microvascular hyperpermeability.
@nl
prefLabel
Interaction of PKC and NOS in signal transduction of microvascular hyperpermeability.
@en
Interaction of PKC and NOS in signal transduction of microvascular hyperpermeability.
@nl
P1476
Interaction of PKC and NOS in signal transduction of microvascular hyperpermeability.
@en
P304
P407
P433
P577
1997-11-01T00:00:00Z