Protein kinase C-epsilon modulates mitochondrial function and active Na+ transport after oxidant injury in renal cells.
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No oxygen? No problem! Intrinsic brain tolerance to hypoxia in vertebrates.Reactive oxygen species and dopamine receptor function in essential hypertensionProtein kinase C-epsilon activation induces mitochondrial dysfunction and fragmentation in renal proximal tubulesγ-Tocotrienol protects against mitochondrial dysfunction and renal cell death.Neuroprotection: lessons from hibernators.Activation of ERK1/2 pathway mediates oxidant-induced decreases in mitochondrial function in renal cells.Protein kinase C-α activation promotes recovery of mitochondrial function and cell survival following oxidant injury in renal cells.Succinate ameliorates energy deficits and prevents dysfunction of complex I in injured renal proximal tubular cellsAkt activation improves oxidative phosphorylation in renal proximal tubular cells following nephrotoxicant injuryVascular endothelial cells mediate mechanical stimulation-induced enhancement of endothelin hyperalgesia via activation of P2X2/3 receptors on nociceptors.Recent advances in renal hypoxia: insights from bench experiments and computer simulations.Protein kinase C-α interaction with iHSP70 in mitochondria promotes recovery of mitochondrial function after injury in renal proximal tubular cellsIschemic preconditioning targets the respiration of synaptic mitochondria via protein kinase C epsilon.Protein kinase C epsilon activation delays neuronal depolarization during cardiac arrest in the euthermic arctic ground squirrel.Mitochondrial regulation of cell cycle and proliferation.Regulation of cellular gas exchange, oxygen sensing, and metabolic control.Mangiferin attenuates oxidative stress induced renal cell damage through activation of PI3K induced Akt and Nrf-2 mediated signaling pathways.Mitochondrial biogenesis restores oxidative metabolism during Staphylococcus aureus sepsis.Renal PKC-ε deficiency attenuates acute kidney injury and ischemic allograft injury via TNF-α-dependent inhibition of apoptosis and inflammation.Deletion of protein kinase C-ε attenuates mitochondrial dysfunction and ameliorates ischemic renal injury.An Orally Active Epac Inhibitor Reverses Mechanical Allodynia and Loss of Intraepidermal Nerve Fibers \iIn A Mouse Model of Chemotherapy-Induced Peripheral Neuropathy.Elevated levels of DNA repair enzymes and antioxidative enzymes by (+)-catechin in murine microglia cells after oxidative stress.
P2860
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P2860
Protein kinase C-epsilon modulates mitochondrial function and active Na+ transport after oxidant injury in renal cells.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Protein kinase C-epsilon modul ...... oxidant injury in renal cells.
@en
Protein kinase C-epsilon modul ...... oxidant injury in renal cells.
@nl
type
label
Protein kinase C-epsilon modul ...... oxidant injury in renal cells.
@en
Protein kinase C-epsilon modul ...... oxidant injury in renal cells.
@nl
prefLabel
Protein kinase C-epsilon modul ...... oxidant injury in renal cells.
@en
Protein kinase C-epsilon modul ...... oxidant injury in renal cells.
@nl
P2093
P2860
P1476
Protein kinase C-epsilon modul ...... oxidant injury in renal cells.
@en
P2093
Diana Bakajsova
Ginger L Clifton
Grazyna Nowak
P2860
P304
P356
10.1152/AJPRENAL.00275.2003
P577
2003-10-21T00:00:00Z