Mechanisms through which high glucose concentration raises [Ca2+]i in renal proximal tubular cells.
about
Calcium mediates high glucose-induced HIF-1α and VEGF expression in cultured rat retinal Müller cells through CaMKII-CREB pathway.New cardioprotective agent K201 is natriuretic and glomerular filtration rate enhancing.Urea signaling to ERK phosphorylation in renal medullary cells requires extracellular calcium but not calcium entry.The amino-terminal domains of the ezrin, radixin, and moesin (ERM) proteins bind advanced glycation end products, an interaction that may play a role in the development of diabetic complications.Gliclazide attenuates the intracellular Ca2+ changes induced in vitro by ischemia in the retinal slices of rats with streptozotocin-induced diabetes.
P2860
Mechanisms through which high glucose concentration raises [Ca2+]i in renal proximal tubular cells.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
Mechanisms through which high ...... renal proximal tubular cells.
@en
Mechanisms through which high ...... renal proximal tubular cells.
@nl
type
label
Mechanisms through which high ...... renal proximal tubular cells.
@en
Mechanisms through which high ...... renal proximal tubular cells.
@nl
prefLabel
Mechanisms through which high ...... renal proximal tubular cells.
@en
Mechanisms through which high ...... renal proximal tubular cells.
@nl
P2093
P1433
P1476
Mechanisms through which high ...... renal proximal tubular cells.
@en
P2093
Marcinkowski W
Smogorzewski M
Symonian M
P304
P356
10.1046/J.1523-1755.1998.00109.X
P407
P577
1998-10-01T00:00:00Z
P5875
P6179
1028494709