Defective calcium handling in cardiomyocytes isolated from hearts subjected to ischemia-reperfusion.
about
Hearts from mice fed a non-obesogenic high-fat diet exhibit changes in their oxidative state, calcium and mitochondria in parallel with increased susceptibility to reperfusion injuryIschemia-reperfusion-induced changes in sarcolemmal Na+/K+-ATPase are due to the activation of calpain in the heartRiluzole protects against cardiac ischaemia and reperfusion damage via block of the persistent sodium current.Polyol pathway impairs the function of SERCA and RyR in ischemic-reperfused rat hearts by increasing oxidative modifications of these proteins.Cardioplegic strategies to protect the hypertrophic heart during cardiac surgery.Cardiac hypertrophy, substrate utilization and metabolic remodelling: cause or effect?The cardiac persistent sodium current: an appealing therapeutic target?N-Methyl-D-Aspartate Receptor (NMDAR)-Driven Calcium Influx Potentiates the Adverse Effects of Myocardial Ischemia-Reperfusion Injury Ex Vivo.Oestrogen changed cardiomyocyte contraction and beta-adrenoceptor expression in rat hearts subjected to ischaemia-reperfusion.Attenuation of ischemia-reperfusion-induced alterations in intracellular Ca2+ in cardiomyocytes from hearts treated with N-acetylcysteine and N-mercaptopropionylglycine.Involvement of sarcoplasmic reticulum in changing intracellular calcium due to Na+/K+-ATPase inhibition in cardiomyocytes.Attenuation of extracellular ATP response in cardiomyocytes isolated from hearts subjected to ischemia-reperfusion.Modification of intracellular calcium concentration in cardiomyocytes by inhibition of sarcolemmal Na+/H+ exchanger.Sarcolemmal cation channels and exchangers modify the increase in intracellular calcium in cardiomyocytes on inhibiting Na+-K+-ATPase.Mechanisms of cardiodepression by an Na+-H+ exchange inhibitor methyl-N-isobutyl amiloride (MIA) on the heart: lack of beneficial effects in ischemia-reperfusion injury.Modification of Ca(2+)-handling in cardiomyocytes by redox sensitive mechanisms in response to ouabain.beta2-Adrenoceptor confers cardioprotection against hypoxia in isolated ventricular myocytes and the effects depend on estrogenic environment.Mitochondrial oxidative phosphorylation in hearts subjected to Ca2+ depletion and Ca2+ repletion.Strategies for the regulation of intracellular calcium in ischemic heart disease.Role of oxidative stress in ischemia-reperfusion-induced alterations in myofibrillar ATPase activities and gene expression in the heart.Differences in ischemia-reperfusion-induced endothelial changes in hearts perfused at constant flow and constant pressure.Modification of alterations in cardiac function and sarcoplasmic reticulum by vanadate in ischemic-reperfused rat hearts.Involvement of Na+/Ca2+ exchanger in catecholamine-induced increase in intracellular calcium in cardiomyocytes.Increased mitochondrial calcium coexists with decreased reperfusion injury in postconditioned (but not preconditioned) hearts.HDAC Inhibition Improves the Sarcoendoplasmic Reticulum Ca2+-ATPase Activity in Cardiac Myocytes.Abolition of reperfusion-induced arrhythmias in hearts from thiamine-deficient rats.Activation of proteolytic enzymes and depression of the sarcolemmal Na+/K+-ATPase in ischemia-reperfused heart may be mediated through oxidative stress.Effects of phosphodiestrase type 5 inhibitors in epinephrine-induced arrhythmia in rats: Involvement of lactate dehydrogenase and creatine kinase downregulation and adiponectin expression.Expression of SERCA isoform with faster Ca2+ transport properties improves postischemic cardiac function and Ca2+ handling and decreases myocardial infarction.
P2860
Q28388331-41DA8100-162B-451D-970E-BA0ADFD3BAE9Q28583177-C294E373-3CA7-4BD9-8A0D-382C3B9E475CQ34112946-0C6422F7-DA86-4DE7-87BF-3DDE6BEFAEBBQ34595686-AF5EB468-7DC3-4425-ACFE-FC47BB5A216EQ35737828-5319CA93-1720-4078-928E-7E20BFBE9BF6Q36381499-B21DC94A-C698-4120-8630-17C47AA2AC3DQ37028768-ABC40DFD-3706-4016-9B15-5605AE9316A1Q38641093-B6DF1101-9E7D-4071-9939-52F228F08E30Q42435879-363FC648-A588-442C-B764-E159B210F1FCQ42464816-D50FB26D-235F-4769-8E2D-10B97B3F2A89Q42474745-320F106F-B5FB-4DDB-974C-CE7AF0A8E1A0Q42476762-6034E87F-342E-4F63-959E-B3EB2AC1EAC2Q42498622-453FBAE8-E949-414F-8963-1EBC68442768Q42508830-0A327022-1B8A-4C8A-86DD-49055C76442CQ42512311-6B06352B-6323-45CF-82F9-062B559BD848Q42520123-FD8A6F66-94BB-4492-B658-95561B0FCF27Q42985634-FB1EE920-3E50-4616-A8D7-72A597ED5F85Q43246057-FF335896-0788-4D6E-8FEC-89E302806D75Q45168252-5716AE72-C8FE-4186-8D8D-DC535778392AQ46113689-AC5EAFB6-EFC6-4C15-B084-E5B7272EC6BAQ46333829-F46DEED1-D307-4E8E-91F9-4196B548CC3FQ46477502-131BE2E3-F2CE-4EE7-B41B-A629E15EDFB6Q46697059-7FB02EB2-FF24-4012-9269-CBC46CD76B3DQ46939197-F6C029B4-B188-47E3-8289-F5CE376F7579Q48134207-710F7CF6-2CA8-4769-A3B9-0809FFED171BQ48236424-2D58F3E5-3840-4386-933D-CB17EF6FE29CQ48735973-5C7ED613-6041-43D0-91A6-0EA2B155D1BAQ49892684-DE74201D-8E9D-4435-AD84-6B337BE6E1A5Q53540868-1D40BA32-B507-4769-969E-DABA43D85DD4
P2860
Defective calcium handling in cardiomyocytes isolated from hearts subjected to ischemia-reperfusion.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Defective calcium handling in ...... ected to ischemia-reperfusion.
@en
Defective calcium handling in ...... ected to ischemia-reperfusion.
@nl
type
label
Defective calcium handling in ...... ected to ischemia-reperfusion.
@en
Defective calcium handling in ...... ected to ischemia-reperfusion.
@nl
prefLabel
Defective calcium handling in ...... ected to ischemia-reperfusion.
@en
Defective calcium handling in ...... ected to ischemia-reperfusion.
@nl
P2860
P1476
Defective calcium handling in ...... ected to ischemia-reperfusion.
@en
P2093
Harjot K Saini
Naranjan S Dhalla
P2860
P304
P356
10.1152/AJPHEART.01153.2004
P577
2005-01-14T00:00:00Z