Acute hypoxia increases intracellular [Ca2+] in pulmonary arterial smooth muscle by enhancing capacitative Ca2+ entry.
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Bone morphogenetic protein 4 enhances canonical transient receptor potential expression, store-operated Ca2+ entry, and basal [Ca2+]i in rat distal pulmonary arterial smooth muscle cellsAcute oxygen-sensing mechanisms.The Role of Transient Receptor Potential Channel 6 Channels in the Pulmonary VasculatureROS-dependent signaling mechanisms for hypoxic Ca(2+) responses in pulmonary artery myocytes.NOX4 mediates BMP4-induced upregulation of TRPC1 and 6 protein expressions in distal pulmonary arterial smooth muscle cellsA randomly-controlled study on the cardiac function at the early stage of return to the plains after short-term exposure to high altitude.High altitude pulmonary hypertension: role of K+ and Ca2+ channels.Interactions between calcium and reactive oxygen species in pulmonary arterial smooth muscle responses to hypoxia.Classical transient receptor potential channel 6 (TRPC6) is essential for hypoxic pulmonary vasoconstriction and alveolar gas exchangeHypoxia. 4. Hypoxia and ion channel functionTRPC channels in smooth muscle cellsEnhancement of myofilament calcium sensitivity by acute hypoxia in rat distal pulmonary arteriesBMP4 increases the expression of TRPC and basal [Ca2+]i via the p38MAPK and ERK1/2 pathways independent of BMPRII in PASMCsCa2+ responses of pulmonary arterial myocytes to acute hypoxia require release from ryanodine and inositol trisphosphate receptors in sarcoplasmic reticulum.Hypoxia-induced migration in pulmonary arterial smooth muscle cells requires calcium-dependent upregulation of aquaporin 1Hypoxic pulmonary vasoconstriction: mechanisms and controversies.AMP-activated protein kinase and the regulation of Ca2+ signalling in O2-sensing cells.AMP-activated protein kinase underpins hypoxic pulmonary vasoconstriction and carotid body excitation by hypoxia in mammals.Effect of chronic perinatal hypoxia on the role of rho-kinase in pulmonary artery contraction in newborn lambs.Sodium tanshinone IIA sulfonate inhibits canonical transient receptor potential expression in pulmonary arterial smooth muscle from pulmonary hypertensive rats.Differences in STIM1 and TRPC expression in proximal and distal pulmonary arterial smooth muscle are associated with differences in Ca2+ responses to hypoxiaBone morphogenetic protein 2 decreases TRPC expression, store-operated Ca(2+) entry, and basal [Ca(2+)]i in rat distal pulmonary arterial smooth muscle cells.Effects of distension on airway inflammation and venular P-selectin expressionThe non-excitable smooth muscle: calcium signaling and phenotypic switching during vascular disease.Sodium tanshinone IIA sulfonate inhibits hypoxia-induced enhancement of SOCE in pulmonary arterial smooth muscle cells via the PKG-PPAR-γ signaling axisMechanisms of oxygen sensing: a key to therapy of pulmonary hypertension and patent ductus arteriosus.Knockdown of stromal interaction molecule 1 attenuates store-operated Ca2+ entry and Ca2+ responses to acute hypoxia in pulmonary arterial smooth muscle.Acute hypoxia activates store-operated Ca(2+) entry and increases intracellular Ca(2+) concentration in rat distal pulmonary venous smooth muscle cellsSildenafil inhibits hypoxia-induced transient receptor potential canonical protein expression in pulmonary arterial smooth muscle via cGMP-PKG-PPARγ axisRole of ASIC1 in the development of chronic hypoxia-induced pulmonary hypertension.TRP channels.Hypoxia-dependent reactive oxygen species signaling in the pulmonary circulation: focus on ion channels.NADPH oxidases-do they play a role in TRPC regulation under hypoxia?Lung Circulation.Role of phosphatase and tensin homolog in hypoxic pulmonary vasoconstriction.Hypoxia increases ROS signaling and cytosolic Ca(2+) in pulmonary artery smooth muscle cells of mouse lungs slices.Role of ROS signaling in differential hypoxic Ca2+ and contractile responses in pulmonary and systemic vascular smooth muscle cells.Chloroquine is a potent pulmonary vasodilator that attenuates hypoxia-induced pulmonary hypertension.Mitochondrial ROS-PKCepsilon signaling axis is uniquely involved in hypoxic increase in [Ca2+]i in pulmonary artery smooth muscle cells.Role of InsP3 and ryanodine receptors in the activation of capacitative Ca2+ entry by store depletion or hypoxia in canine pulmonary arterial smooth muscle cells.
P2860
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P2860
Acute hypoxia increases intracellular [Ca2+] in pulmonary arterial smooth muscle by enhancing capacitative Ca2+ entry.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Acute hypoxia increases intrac ...... ncing capacitative Ca2+ entry.
@en
Acute hypoxia increases intrac ...... ncing capacitative Ca2+ entry.
@nl
type
label
Acute hypoxia increases intrac ...... ncing capacitative Ca2+ entry.
@en
Acute hypoxia increases intrac ...... ncing capacitative Ca2+ entry.
@nl
prefLabel
Acute hypoxia increases intrac ...... ncing capacitative Ca2+ entry.
@en
Acute hypoxia increases intrac ...... ncing capacitative Ca2+ entry.
@nl
P2093
P2860
P1476
Acute hypoxia increases intrac ...... ncing capacitative Ca2+ entry.
@en
P2093
J T Sylvester
Larissa A Shimoda
Letitia Weigand
Wenqian Wang
P2860
P304
P356
10.1152/AJPLUNG.00448.2004
P577
2005-01-21T00:00:00Z