ACE-inhibition attenuates cardiac cell damage and preserves release of NO in the postischemic heart.
about
Nitric oxide homeostasis as a target for drug additives to cardioplegiaKallidin-like peptide mediates the cardioprotective effect of the ACE inhibitor captopril against ischaemic reperfusion injury of rat heart.Exogenous nitric oxide requires an endothelial glycocalyx to prevent postischemic coronary vascular leak in guinea pig hearts.
P2860
ACE-inhibition attenuates cardiac cell damage and preserves release of NO in the postischemic heart.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
ACE-inhibition attenuates card ...... NO in the postischemic heart.
@en
ACE-inhibition attenuates card ...... NO in the postischemic heart.
@nl
type
label
ACE-inhibition attenuates card ...... NO in the postischemic heart.
@en
ACE-inhibition attenuates card ...... NO in the postischemic heart.
@nl
prefLabel
ACE-inhibition attenuates card ...... NO in the postischemic heart.
@en
ACE-inhibition attenuates card ...... NO in the postischemic heart.
@nl
P1433
P1476
ACE-inhibition attenuates card ...... NO in the postischemic heart.
@en
P2093
P356
10.1016/S0162-3109(99)00108-3
P577
1999-10-01T00:00:00Z