Effect of angiotensin II-induced arterial hypertension on the voltage-dependent contractions of mouse arteries.

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Isometric Stretch Alters Vascular Reactivity of Mouse Aortic Segments.Short-Term Angiotensin II Treatment Affects Large Artery Biomechanics and Function in the Absence of Small Artery Alterations in Mice.

P2860

Q37703018-B8438789-D95C-4134-A7FB-264D145A07E5 Q54951106-B5704F43-AA27-4E42-AAC3-F9B539C7C6A6

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Effect of angiotensin II-induced arterial hypertension on the voltage-dependent contractions of mouse arteries.

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http://www.wikidata.org/entity/Q42483189

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

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2015年論文

@zh-tw

2015年论文

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2015年论文

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2015年论文

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name

Effect of angiotensin II-induc ...... ontractions of mouse arteries.

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Effect of angiotensin II-induc ...... ontractions of mouse arteries.

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type

Item

label

Effect of angiotensin II-induc ...... ontractions of mouse arteries.

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Effect of angiotensin II-induc ...... ontractions of mouse arteries.

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Effect of angiotensin II-induc ...... ontractions of mouse arteries.

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Effect of angiotensin II-induc ...... ontractions of mouse arteries.

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P1476

Effect of angiotensin II-induc ...... ontractions of mouse arteries.

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P2093

Arthur J A Leloup

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Cor E Van Hove

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Guido R Y De Meyer

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Paul Fransen

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P2860

Dominant role of smooth muscle L-type calcium channel Cav1.2 for blood pressure regulation

Signature combinatorial splicing profiles of rat cardiac- and smooth-muscle Cav1.2 channels with distinct biophysical properties.

Atherosclerosis-related molecular alteration of the human CaV1.2 calcium channel alpha1C subunit.

Smooth muscle-selective alternatively spliced exon generates functional variation in Cav1.2 calcium channels.

Pulse pressure, endothelium function, and arterial stiffness in spontaneously hypertensive rats.

Mechanisms by which angiotensin-converting enzyme inhibitors prevent diabetes and cardiovascular disease.

Contribution of transient and sustained calcium influx, and sensitization to depolarization-induced contractions of the intact mouse aorta

Vascular Smooth Muscle Cell Signaling Mechanisms for Contraction to Angiotensin II and Endothelin-1.

Vasodilator efficacy of nitric oxide depends on mechanisms of intracellular calcium mobilization in mouse aortic smooth muscle cells.

Augmented S-nitrosylation contributes to impaired relaxation in angiotensin II hypertensive mouse aorta: role of thioredoxin reductase.

P2888

s00424-015-1737-x

P304

257-267

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scholarly article

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10.1007/S00424-015-1737-X

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P478

468

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Gilles W De Keulenaer

Dorien M Schrijvers

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2015-10-03T00:00:00Z

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282570076

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26432297

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Effect of angiotensin II-induced arterial hypertension on the voltage-dependent contractions of mouse arteries.

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P2860

P2860

Effect of angiotensin II-induced arterial hypertension on the voltage-dependent contractions of mouse arteries.

description

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P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P433

P478

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P5875

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P1476

P2093

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P2888

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P356

P433

P478

P50

P577

P5875

P698