Lack of the DNA repair enzyme OGG1 sensitizes dopamine neurons to manganese toxicity during development.
about
8-Oxoguanine DNA glycosylase (OGG1) deficiency increases susceptibility to obesity and metabolic dysfunctionOgg1 null mice exhibit age-associated loss of the nigrostriatal pathway and increased sensitivity to MPTP.8-Oxoguanine-DNA glycosylase 1 deficiency modifies allergic airway inflammation by regulating STAT6 and IL-4 in cells and in mice.Brain capacity for repair of oxidatively damaged DNA and preservation of neuronal function.Oxidative DNA damage in disease--insights gained from base excision repair glycosylase-deficient mouse models.8-oxoguanine DNA glycosylase (OGG1) deficiency elicits coordinated changes in lipid and mitochondrial metabolism in muscle.
P2860
Lack of the DNA repair enzyme OGG1 sensitizes dopamine neurons to manganese toxicity during development.
description
2005 nî lūn-bûn
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2005年の論文
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2005年論文
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2005年論文
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2005年論文
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2005年論文
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2005年論文
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2005年论文
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2005年论文
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name
Lack of the DNA repair enzyme ...... e toxicity during development.
@en
Lack of the DNA repair enzyme ...... e toxicity during development.
@nl
type
label
Lack of the DNA repair enzyme ...... e toxicity during development.
@en
Lack of the DNA repair enzyme ...... e toxicity during development.
@nl
prefLabel
Lack of the DNA repair enzyme ...... e toxicity during development.
@en
Lack of the DNA repair enzyme ...... e toxicity during development.
@nl
P2093
P2860
P1433
P1476
Lack of the DNA repair enzyme ...... e toxicity during development.
@en
P2093
Celeste Bolin
David P Cox
Fernando Cardozo-Pelaez
P2860
P304
P356
10.3727/000000005783992007
P577
2005-01-01T00:00:00Z