Ras activation in response to phorbol ester proceeds independently of the EGFR via an unconventional nucleotide-exchange factor system in COS-7 cells.
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Impaired TrkB-mediated ERK1/2 activation in huntington disease knock-in striatal cells involves reduced p52/p46 Shc expressionRegulation of ras exchange factors and cellular localization of ras activation by lipid messengers in T cells.Ras palmitoylation is necessary for N-Ras activation and signal propagation in growth factor signalling.Down-regulation of c-Cbl by morphine accounts for persistent ERK1/2 signaling in delta-opioid receptor-expressing HEK293 cellsRasGRP1 overexpression in T-ALL increases basal nucleotide exchange on Ras rendering the Ras/PI3K/Akt pathway responsive to protumorigenic cytokines.Phosphorylation and transcriptional activity regulation of retinoid-related orphan receptor alpha 1 by protein kinases C.Lysophosphatidylcholine Drives Neuroblast Cell Fate.JunB defines functional and structural integrity of the epidermo-pilosebaceous unit in the skin
P2860
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P2860
Ras activation in response to phorbol ester proceeds independently of the EGFR via an unconventional nucleotide-exchange factor system in COS-7 cells.
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name
Ras activation in response to ...... factor system in COS-7 cells.
@en
Ras activation in response to ...... factor system in COS-7 cells.
@nl
type
label
Ras activation in response to ...... factor system in COS-7 cells.
@en
Ras activation in response to ...... factor system in COS-7 cells.
@nl
prefLabel
Ras activation in response to ...... factor system in COS-7 cells.
@en
Ras activation in response to ...... factor system in COS-7 cells.
@nl
P2093
P2860
P356
P1433
P1476
Ras activation in response to ...... factor system in COS-7 cells.
@en
P2093
Ignacio Rubio
Katrin Beer
Knut Rennert
Matthias Dürst
Reinhard Wetzker
Stacey L Stang
Ute Wittig
P2860
P304
P356
10.1042/BJ20060160
P407
P577
2006-09-01T00:00:00Z