1,25-dihydroxyvitamin D-responsive element and glucocorticoid repression in the osteocalcin gene.
about
Factors involved in the duodenal expression of the human calbindin-D9k geneTwo mutations in the hormone binding domain of the vitamin D receptor cause tissue resistance to 1,25 dihydroxyvitamin D3In vitro and in vivo evidence for orphan nuclear receptor RORalpha function in bone metabolismMolecular analysis of the inhibition of interleukin-8 production by dexamethasone in a human fibrosarcoma cell line1 alpha,25-(OH)2 vitamin D3 enhances expression of the genes encoding Ca(2+)-binding proteins MRP-8 and MRP-14Prolactin blocks nuclear translocation of VDR by regulating its interaction with BRCA1 in osteosarcoma cellsOsteocrin, a novel bone-specific secreted protein that modulates the osteoblast phenotypeVitamin D and the Pathogenesis of Inflammatory Bowel Disease.Regulation of human osteocalcin promoter in hormone-independent human prostate cancer cells.Involvement of SMRT corepressor in transcriptional repression by the vitamin D receptorTherapeutic Implications of PPARgamma in Human Osteosarcoma.1 alpha,25-dihydroxyvitamin D3 regulates the expression of carbonic anhydrase II in nonerythroid avian bone marrow cells.Coordinate occupancy of AP-1 sites in the vitamin D-responsive and CCAAT box elements by Fos-Jun in the osteocalcin gene: model for phenotype suppression of transcription.Identification of a DNA sequence responsible for binding of the 1,25-dihydroxyvitamin D3 receptor and 1,25-dihydroxyvitamin D3 enhancement of mouse secreted phosphoprotein 1 (SPP-1 or osteopontin) gene expression.RARs and RXRs: evidence for two autonomous transactivation functions (AF-1 and AF-2) and heterodimerization in vivo.Histone acetylation in vivo at the osteocalcin locus is functionally linked to vitamin D-dependent, bone tissue-specific transcription.1,25-dihydroxyvitamin D3 modulates growth of vascular smooth muscle cells.A bisphosphonate that does not affect osteoclasts prevents osteoblast and osteocyte apoptosis and the loss of bone strength induced by glucocorticoids in mice.Vitamin D: direct effects of vitamin D metabolites on bone: lessons from genetically modified mice.Gene expression profiles resulting from stable loss of p53 mirrors its role in tissue differentiation.Gene-modified adult stem cells regenerate vertebral bone defect in a rat model.Regulation of parathyroid hormone-like peptide in cultured normal human keratinocytes. Effect of growth factors and 1,25 dihydroxyvitamin D3 on gene expression and secretion.p53 and MDM2 are involved in the regulation of osteocalcin gene expression.A composite intragenic silencer domain exhibits negative and positive transcriptional control of the bone-specific osteocalcin gene: promoter and cell type requirements.YY1 regulates vitamin D receptor/retinoid X receptor mediated transactivation of the vitamin D responsive osteocalcin gene.Transcriptional control of the tissue-specific, developmentally regulated osteocalcin gene requires a binding motif for the Msx family of homeodomain proteinsConcepts of osteoblast growth and differentiation: basis for modulation of bone cell development and tissue formation.Associations of vitamin D deficiency and vitamin D receptor (Cdx-2, Fok I, Bsm I and Taq I) polymorphisms with the risk of primary open-angle glaucoma.Postproliferative transcription of the rat osteocalcin gene is reflected by vitamin D-responsive developmental modifications in protein-DNA interactions at basal and enhancer promoter elements.Generation of VDR Knock-Out Mice via Zygote Injection of CRISPR/Cas9 System.Demographics and characterization of 10,282 Randall plaque-related kidney stones: a new epidemic?Constitutive transcription of the osteocalcin gene in osteosarcoma cells is reflected by altered protein-DNA interactions at promoter regulatory elements.Identification of a DNA sequence involved in osteoblast-specific gene expression via interaction with helix-loop-helix (HLH)-type transcription factorsImmunosteroid as a regulator for Th1/Th2 balance: its possible role in autoimmune diseases.New and improved glucocorticoid receptor ligands.1,25-Dihydroxyvitamin D3 suppresses telomerase expression and human cancer growth through microRNA-498.Natural vitamin D3 response elements formed by inverted palindromes: polarity-directed ligand sensitivity of vitamin D3 receptor-retinoid X receptor heterodimer-mediated transactivation.Two distinct osteoblast-specific cis-acting elements control expression of a mouse osteocalcin gene.Development of the osteoblast phenotype: molecular mechanisms mediating osteoblast growth and differentiation.Characterization of a tumor necrosis factor-responsive element which down-regulates the human osteocalcin gene.
P2860
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P2860
1,25-dihydroxyvitamin D-responsive element and glucocorticoid repression in the osteocalcin gene.
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年学术文章
@wuu
1989年学术文章
@zh-cn
1989年学术文章
@zh-hans
1989年学术文章
@zh-my
1989年学术文章
@zh-sg
1989年學術文章
@yue
1989年學術文章
@zh
1989年學術文章
@zh-hant
name
1,25-dihydroxyvitamin D-respon ...... ssion in the osteocalcin gene.
@en
1,25-dihydroxyvitamin D-respon ...... ssion in the osteocalcin gene.
@nl
type
label
1,25-dihydroxyvitamin D-respon ...... ssion in the osteocalcin gene.
@en
1,25-dihydroxyvitamin D-respon ...... ssion in the osteocalcin gene.
@nl
prefLabel
1,25-dihydroxyvitamin D-respon ...... ssion in the osteocalcin gene.
@en
1,25-dihydroxyvitamin D-respon ...... ssion in the osteocalcin gene.
@nl
P2093
P356
P1433
P1476
1,25-dihydroxyvitamin D-respon ...... ssion in the osteocalcin gene.
@en
P2093
P304
P356
10.1126/SCIENCE.2588000
P407
P577
1989-12-01T00:00:00Z