Sustained activation of the Raf/MEK/Erk pathway in response to EGF in stable cell lines expressing the Hepatitis C Virus (HCV) core protein. - Wikidata - awgldk - TriplyDB

Sustained activation of the Raf/MEK/Erk pathway in response to EGF in stable cell lines expressing the Hepatitis C Virus (HCV) core protein.

Sustained activation of the Raf/MEK/Erk pathway in response to EGF in stable cell lines expressing the Hepatitis C Virus (HCV) core protein.

http://www.wikidata.org/entity/Q42506244

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Hepatocellular carcinoma: Will novel targeted drugs really impact the next future?Targeted therapy for hepatocellular carcinoma: novel agents on the horizon Hepatitis C virus core protein promotes proliferation of human hepatoma cells through enhancement of transforming growth factor expression via activation of nuclear factor- B Hepatocellular Carcinoma: Therapeutic Guidelines and Medical Treatment Evolving role of Sorafenib in the management of hepatocellular carcinoma.Relationship of ethnicity and overall survival in patients treated with sorafenib for advanced hepatocellular carcinoma.Erlotinib derivative inhibits hepatocellular carcinoma by targeting CIP2A to reactivate protein phosphatase 2A.Coxsackievirus B3 replication is reduced by inhibition of the extracellular signal-regulated kinase (ERK) signaling pathway.Hepatitis C virus biology.Molecular targets and oxidative stress biomarkers in hepatocellular carcinoma: an overview.Novel insights into the function and dynamics of extracellular matrix in liver fibrosis.Activation of the Ras/Raf/MEK pathway facilitates hepatitis C virus replication via attenuation of the interferon-JAK-STAT pathway.MEK1/2 inhibitors block basal and transforming growth factor 1beta1-stimulated JC virus multiplication.Hepatitis B- and hepatitis C-related hepatocellular carcinomas in the United States: similarities and differences Hepatitis C virus-related hepatocellular carcinoma: An insight into molecular mechanisms and therapeutic strategies Sorafenib for treatment of hepatocellular carcinoma: a systematic review.The hepatitis E virus open reading frame 3 protein activates ERK through binding and inhibition of the MAPK phosphatase Mutant p53-R273H gains new function in sustained activation of EGFR signaling via suppressing miR-27a expression ERK signaling is triggered by hepatitis C virus E2 protein through DC-SIGN Current management of advanced hepatocellular carcinoma RNA viruses and the mitogenic Raf/MEK/ERK signal transduction cascade.Molecular alterations in hepatocellular carcinoma associated with hepatitis B and hepatitis C infections.Phase 2 study of bevacizumab plus erlotinib in patients with advanced hepatocellular cancer Predictors of survival in patients with established cirrhosis and hepatocellular carcinoma treated with sorafenib.Targeted therapy of hepatocellular cancer.Tyrosine kinase inhibitors to treat liver cancer.Emerging MEK inhibitors.Lactate dehydrogenase is a prognostic indicator in patients with hepatocellular carcinoma treated by sorafenib: results from the real life practice in HBV endemic area Nonoperative therapies for combined modality treatment of hepatocellular cancer: expert consensus statement.Latest developments in targeted therapy for hepatocellular carcinoma.AFP measurement in monitoring treatment response of advanced hepatocellular carcinoma to sorafenib: case report and review of the literature.Treating advanced hepatocellular carcinoma: How to get out of first gear.Hepatocellular carcinoma: systemic therapies and future perspectives.Advanced Hepatocellular Cancer: the Current State of Future Research.Targeting the Ras/Raf/MEK/ERK pathway in hepatocellular carcinoma.Sorafenib for the treatment of unresectable hepatocellular carcinoma.Mitogen-activated protein kinase signalling pathways triggered by the hepatitis C virus envelope protein E2: implications for the prevention of infection.RNA interference effectively inhibits mRNA accumulation and protein expression of hepatitis C virus core and E2 genes in human cells.Role of p38 MAPK and RNA-dependent protein kinase (PKR) in hepatitis C virus core-dependent nuclear delocalization of cyclin B1.The vaccinia virus-stimulated mitogen-activated protein kinase (MAPK) pathway is required for virus multiplication.

P2860

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P2860

Sustained activation of the Raf/MEK/Erk pathway in response to EGF in stable cell lines expressing the Hepatitis C Virus (HCV) core protein.

http://www.wikidata.org/entity/Q42506244

description

2001 nî lūn-bûn

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2001年の論文

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2001年学术文章

@wuu

2001年学术文章

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2001年学术文章

@zh-hans

2001年学术文章

@zh-my

2001年学术文章

@zh-sg

2001年學術文章

@yue

2001年學術文章

@zh

2001年學術文章

@zh-hant

name

Sustained activation of the Ra ...... is C Virus (HCV) core protein.

@en

Sustained activation of the Ra ...... pressing the Hepatitis C Virus

@nl

type

label

Sustained activation of the Ra ...... is C Virus (HCV) core protein.

@en

Sustained activation of the Ra ...... pressing the Hepatitis C Virus

@nl

prefLabel

Sustained activation of the Ra ...... is C Virus (HCV) core protein.

@en

Sustained activation of the Ra ...... pressing the Hepatitis C Virus

@nl

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Sustained activation of the Ra ...... tis C Virus (HCV) core protein

@en

P2093

Balsano C

http://www.w3.org/2001/XMLSchema#string

Covone F

http://www.w3.org/2001/XMLSchema#string

Giambartolomei S

http://www.w3.org/2001/XMLSchema#string

P2860

Hepatitis C virus core protein binds to a DEAD box RNA helicase

Reduced MAP kinase phosphatase-1 degradation after p42/p44MAPK-dependent phosphorylation

Hepatitis C virus core protein interacts with 14-3-3 protein and activates the kinase Raf-1

Hepatitis C virus core protein-induced loss of LZIP function correlates with cellular transformation

Hepatitis C virus core protein shows a cytoplasmic localization and associates to cellular lipid storage droplets

The complexity of Raf-1 regulation

The core protein of hepatitis C virus induces hepatocellular carcinoma in transgenic mice

Mitogen-activated protein kinases: specific messages from ubiquitous messengers

Organization and regulation of mitogen-activated protein kinase signaling pathways.

Hepatitis C virus and hepatocarcinogenesis.

P2888

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2606-2610

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scholarly article

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10.1038/SJ.ONC.1204372

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20

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20

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Massimo Levrero

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2001-05-01T00:00:00Z

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11919110

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11420671

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