Transforming growth factor-beta stimulates parathyroid hormone-related protein and osteolytic metastases via Smad and mitogen-activated protein kinase signaling pathways. - Wikidata - awgldk - TriplyDB

Transforming growth factor-beta stimulates parathyroid hormone-related protein and osteolytic metastases via Smad and mitogen-activated protein kinase signaling pathways.

Transforming growth factor-beta stimulates parathyroid hormone-related protein and osteolytic metastases via Smad and mitogen-activated protein kinase signaling pathways.

http://www.wikidata.org/entity/Q42520359

about

Hypoxia and TGF-beta drive breast cancer bone metastases through parallel signaling pathways in tumor cells and the bone microenvironment.14-3-3ζ turns TGF-β's function from tumor suppressor to metastasis promoter in breast cancer by contextual changes of Smad partners from p53 to Gli2 Breast cancer bone metastasis mediated by the Smad tumor suppressor pathway Granulocyte colony-stimulating factor enhances bone tumor growth in mice in an osteoclast-dependent manner Interfering with TGFbeta-induced Smad3 nuclear accumulation differentially affects TGFbeta-dependent gene expression Suppression of NADPH Oxidase Activity May Slow the Expansion of Osteolytic Bone Metastases From palliative therapy to prolongation of survival: (223)RaCl2 in the treatment of bone metastases Bone Metastasis from Renal Cell Carcinoma Use of Animal Models in Understanding Cancer-induced Bone Pain Molecular alterations that drive breast cancer metastasis to bone At the crossroads: EGFR and PTHrP signaling in cancer-mediated diseases of bone Immune regulation of bone metastasis TGFbeta in Cancer 15-deoxy-δ12,14-prostaglandin j2 inhibits osteolytic breast cancer bone metastasis and estrogen deficiency-induced bone loss Drug development against metastasis-related genes and their pathways: A rationale for cancer therapy Tumor-expressed adrenomedullin accelerates breast cancer bone metastasis.TGF-beta-RI kinase inhibitor SD-208 reduces the development and progression of melanoma bone metastases Advances in treating metastatic bone cancer: summary statement for the First Cambridge Conference ALK1 signalling analysis identifies angiogenesis related genes and reveals disparity between TGF-beta and constitutively active receptor induced gene expression Bone-in-culture array as a platform to model early-stage bone metastases and discover anti-metastasis therapies PTHrP gene expression in cancer: do all paths lead to Ets?Matrix rigidity induces osteolytic gene expression of metastatic breast cancer cells.Targeting the Transforming Growth Factor-beta pathway inhibits human basal-like breast cancer metastasis.SMAD4 is a potential prognostic marker in human breast carcinomas.Notch3 in human breast cancer cell lines regulates osteoblast-cancer cell interactions and osteolytic bone metastasis.Discovery of multi-dimensional modules by integrative analysis of cancer genomic data.Integrins and bone metastasis: integrating tumor cell and stromal cell interactions.TGF-beta antagonists: why suppress a tumor suppressor?TGF-β signaling in breast cancer cell invasion and bone metastasis.A KLF4-miRNA-206 autoregulatory feedback loop can promote or inhibit protein translation depending upon cell context.Phase I study of GC1008 (fresolimumab): a human anti-transforming growth factor-beta (TGFβ) monoclonal antibody in patients with advanced malignant melanoma or renal cell carcinoma.TAK1-TAB2 signaling contributes to bone destruction by breast carcinoma cells The two faces of transforming growth factor beta in carcinogenesis Transforming growth factor beta-activated kinase 1 is a key mediator of ovine follicle-stimulating hormone beta-subunit expression.Bone-immune cell crosstalk: bone diseases.Homing of cancer cells to the bone TGF-β Induces Degradation of PTHrP Through Ubiquitin-Proteasome System in Hepatocellular Carcinoma.HGF and TGFβ1 differently influenced Wwox regulatory function on Twist program for mesenchymal-epithelial transition in bone metastatic versus parental breast carcinoma cells Partners in crime: the TGFβ and MAPK pathways in cancer progression.Effect of transforming growth factor beta (TGF-β) receptor I kinase inhibitor on prostate cancer bone growth.

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P2860

Transforming growth factor-beta stimulates parathyroid hormone-related protein and osteolytic metastases via Smad and mitogen-activated protein kinase signaling pathways.

http://www.wikidata.org/entity/Q42520359

description

2002 nî lūn-bûn

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2002年の論文

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年學術文章

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2002年學術文章

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2002年學術文章

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name

Transforming growth factor-bet ...... ein kinase signaling pathways.

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Transforming growth factor-bet ...... ein kinase signaling pathways.

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type

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Transforming growth factor-bet ...... ein kinase signaling pathways.

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Transforming growth factor-bet ...... ein kinase signaling pathways.

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Transforming growth factor-bet ...... ein kinase signaling pathways.

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Transforming growth factor-bet ...... ein kinase signaling pathways.

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Journal of Biological Chemistry

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Transforming growth factor-bet ...... ein kinase signaling pathways.

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Barry G Grubbs

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John M Chirgwin

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Juan Juan Yin

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Katri S Selander

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Mark Dallas

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Sanna-Maria Kakonen

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Suzanne Burns

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Wayne A Rankin

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Yong Cui

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10.1074/JBC.M202561200

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27

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Theresa A Guise

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2002-04-18T00:00:00Z

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11964407

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