Network-level changes in expression of inducible Fos-Jun proteins in the striatum during chronic cocaine treatment and withdrawal.
about
Amphetamine and pseudoephedrine cross-tolerance measured by c-Fos protein expression in brains of chronically treated ratsAddiction-related gene regulation: risks of exposure to cognitive enhancers vs. other psychostimulantsDeltaFosB: a sustained molecular switch for addictionReview. Transcriptional mechanisms of addiction: role of DeltaFosBDistinct patterns of DeltaFosB induction in brain by drugs of abuseRequirement for the POZ/BTB protein NAC1 in acute but not chronic psychomotor stimulant responseGlucocorticoids regulation of FosB/ΔFosB expression induced by chronic opiate exposure in the brain stress systemC/EBPbeta couples dopamine signalling to substance P precursor gene expression in striatal neuronesStriatal signal transduction and drug addictionQuantitative mapping of cocaine-induced ΔFosB expression in the striatum of male and female ratsCocaine activates Homer1 immediate early gene transcription in the mesocorticolimbic circuit: differential regulation by dopamine and glutamate signalingChronic methamphetamine exposure suppresses the striatal expression of members of multiple families of immediate early genes (IEGs) in the rat: normalization by an acute methamphetamine injectionPancreatic homeodomain transcription factor IDX1/IPF1 expressed in developing brain regulates somatostatin gene transcription in embryonic neural cellsSpatiotemporal pattern of striatal ERK1/2 phosphorylation in a rat model of L-DOPA-induced dyskinesia and the role of dopamine D1 receptors.Time course of striatal DeltaFosB-like immunoreactivity and prodynorphin mRNA levels after discontinuation of chronic dopaminomimetic treatment.Role of the ERK pathway in psychostimulant-induced locomotor sensitization.Overexpression of DeltaFosB is associated with attenuated cocaine-induced suppression of saccharin intake in mice.Relapse to cocaine seeking increases activity-regulated gene expression differentially in the prefrontal cortex of abstinent rats.Assessment of genome and proteome profiles in cocaine abuseMechanisms of HIV Transcriptional Regulation by Drugs of Abuse.A molecular profile of cocaine abuse includes the differential expression of genes that regulate transcription, chromatin, and dopamine cell phenotype.Environmental enrichment during early stages of life reduces the behavioral, neurochemical, and molecular effects of cocaine.CBP in the nucleus accumbens regulates cocaine-induced histone acetylation and is critical for cocaine-associated behaviors.Local circuit neurons in the striatum regulate neural and behavioral responses to dopaminergic stimulationMeCP2 in the nucleus accumbens contributes to neural and behavioral responses to psychostimulantsRats markedly escalate their intake and show a persistent susceptibility to reinstatement only when cocaine is injected rapidly.Regulation of motor performance and striatal function by synaptic scaffolding proteins of the Homer1 familyRegulation of gene expression and cocaine reward by CREB and DeltaFosB.Toward a molecular understanding of psychostimulant actions using genetically engineered dopamine receptor knockout mice as model systems.Cocaine-induced dendritic spine formation in D1 and D2 dopamine receptor-containing medium spiny neurons in nucleus accumbens.Genome-wide profiling identifies a subset of methamphetamine (METH)-induced genes associated with METH-induced increased H4K5Ac binding in the rat striatum.Adenosine A₂A receptors in striatal glutamatergic terminals and GABAergic neurons oppositely modulate psychostimulant action and DARPP-32 phosphorylation.Differential dopamine receptor occupancy underlies L-DOPA-induced dyskinesia in a rat model of Parkinson's diseaseBasal Ganglia disorders associated with imbalances in the striatal striosome and matrix compartments.Differential regulation of MeCP2 phosphorylation in the CNS by dopamine and serotonin.Cocainomics: new insights into the molecular basis of cocaine addiction.Preventive role of social interaction for cocaine conditioned place preference: correlation with FosB/DeltaFosB and pCREB expression in rat mesocorticolimbic areasLevels of neural progenitors in the hippocampus predict memory impairment and relapse to drug seeking as a function of excessive methamphetamine self-administration.Quantitative autoradiographic study on receptor regulation in the basal ganglia in rat model of levodopa-induced motor complicationsNeurobiologic processes in drug reward and addiction.
P2860
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P2860
Network-level changes in expression of inducible Fos-Jun proteins in the striatum during chronic cocaine treatment and withdrawal.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh
1996年學術文章
@zh-hant
name
Network-level changes in expre ...... aine treatment and withdrawal.
@en
Network-level changes in expre ...... aine treatment and withdrawal.
@nl
type
label
Network-level changes in expre ...... aine treatment and withdrawal.
@en
Network-level changes in expre ...... aine treatment and withdrawal.
@nl
prefLabel
Network-level changes in expre ...... aine treatment and withdrawal.
@en
Network-level changes in expre ...... aine treatment and withdrawal.
@nl
P2093
P1433
P1476
Network-level changes in expre ...... aine treatment and withdrawal.
@en
P2093
P304
P356
10.1016/S0896-6273(00)80288-3
P407
P577
1996-07-01T00:00:00Z