Mechanisms of vascular smooth muscle NADPH oxidase 1 (Nox1) contribution to injury-induced neointimal formation. - Wikidata - awgldk - TriplyDB

Mechanisms of vascular smooth muscle NADPH oxidase 1 (Nox1) contribution to injury-induced neointimal formation.

Mechanisms of vascular smooth muscle NADPH oxidase 1 (Nox1) contribution to injury-induced neointimal formation.

http://www.wikidata.org/entity/Q42538641

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Evolution of NADPH Oxidase Inhibitors: Selectivity and Mechanisms for Target Engagement Biochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular system Bang-bang model for regulation of local blood flow Redox signaling in cardiovascular pathophysiology: A focus on hydrogen peroxide and vascular smooth muscle cells The role of oxidative stress and autophagy in atherosclerosis Potential Role of Protein Disulfide Isomerase in Metabolic Syndrome-Derived Platelet Hyperactivity Regulation of signal transduction by reactive oxygen species in the cardiovascular system Oxidases and peroxidases in cardiovascular and lung disease: new concepts in reactive oxygen species signaling Poldip2, a novel regulator of Nox4 and cytoskeletal integrity in vascular smooth muscle cells Angiotensin II enhances AT1-Nox1 binding and stimulates arterial smooth muscle cell migration and proliferation through AT1, Nox1, and interleukin-18.Hu antigen R is required for NOX-1 but not NOX-4 regulation by inflammatory stimuli in vascular smooth muscle cells.NADPH oxidases in vascular pathology.NADPH oxidases: progress and opportunities.Redox control of vascular smooth muscle migration.NADPH oxidases: functions and pathologies in the vasculature.Identification of new autoantibody specificities directed at proteins involved in the transforming growth factor β pathway in patients with systemic sclerosis Coronary endothelial function and vascular smooth muscle proliferation are programmed by early-gestation dexamethasone exposure in sheep.AMPKalpha2 deletion causes aberrant expression and activation of NAD(P)H oxidase and consequent endothelial dysfunction in vivo: role of 26S proteasomes.Upregulation of intermediate-conductance Ca2+-activated K+ channels (KCNN4) in porcine coronary smooth muscle requires NADPH oxidase 5 (NOX5).Upregulation of Nox1 in vascular smooth muscle leads to impaired endothelium-dependent relaxation via eNOS uncoupling.Comparative pharmacology of chemically distinct NADPH oxidase inhibitors.Nox2 is required for macrophage chemotaxis towards CSF-1.Function and design of the Nox1 system in vascular smooth muscle cells.Intestinal NADPH oxidase 2 activity increases in a neonatal rat model of necrotizing enterocolitis.Molecular regulation of NADPH oxidase 5 via the MAPK pathway.Nitric oxide inhibits neointimal hyperplasia following vascular injury via differential, cell-specific modulation of SOD-1 in the arterial wall Role for Nox1 NADPH oxidase in atherosclerosis Positive feedback regulation of proliferation in vascular smooth muscle cells stimulated by lipopolysaccharide is mediated through the TLR 4/Rac1/Akt pathway.Activation of NADPH oxidase 1 increases intracellular calcium and migration of smooth muscle cells.Impact of Nox5 polymorphisms on basal and stimulus-dependent ROS generation.Platelet-derived growth factor (PDGF) regulates Slingshot phosphatase activity via Nox1-dependent auto-dephosphorylation of serine 834 in vascular smooth muscle cells.Increased cerebral oxygen metabolism and ischemic stress in subjects with metabolic syndrome-associated risk factors: preliminary observations.Smooth muscle specific overexpression of p22phox potentiates carotid artery wall thickening in response to injury.Nox1 transactivation of epidermal growth factor receptor promotes N-cadherin shedding and smooth muscle cell migration Redox regulation of cell migration and adhesion Regulation of NAD(P)H oxidases by AMPK in cardiovascular systems Combating oxidative stress in vascular disease: NADPH oxidases as therapeutic targets The NOX toolbox: validating the role of NADPH oxidases in physiology and disease.Increased epidermal growth factor-like ligands are associated with elevated vascular nicotinamide adenine dinucleotide phosphate oxidase in a primate model of atherosclerosis.The role of Nox-mediated oxidation in the regulation of cytoskeletal dynamics

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Mechanisms of vascular smooth muscle NADPH oxidase 1 (Nox1) contribution to injury-induced neointimal formation.

http://www.wikidata.org/entity/Q42538641

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2009年論文

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2009年论文

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2009年论文

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name

Mechanisms of vascular smooth ...... -induced neointimal formation.

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Mechanisms of vascular smooth muscle NADPH oxidase 1

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Mechanisms of vascular smooth ...... -induced neointimal formation.

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Mechanisms of vascular smooth muscle NADPH oxidase 1

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Mechanisms of vascular smooth ...... -induced neointimal formation.

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Mechanisms of vascular smooth muscle NADPH oxidase 1

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ATVBAHA.108.181925

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Arteriosclerosis, Thrombosis, and Vascular Biology

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Mechanisms of vascular smooth ...... -induced neointimal formation.

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Abel Martin Garrido

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Alejandra San Martin

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Anna E Dikalova

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Bernard Lassègue

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Erin Lyons

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J David Lambeth

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Karl-Heinz Krause

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Moo Yeol Lee

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Puja K Mehta

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S Raju Datla

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P2860

Cell transformation by the superoxide-generating oxidase Mox1

Hydrogen peroxide mediates the cell growth and transformation caused by the mitogenic oxidase Nox1

Cell migration: integrating signals from front to back

Novel gp91(phox) homologues in vascular smooth muscle cells : nox1 mediates angiotensin II-induced superoxide formation and redox-sensitive signaling pathways

The mouse Formin mDia1 is a potent actin nucleation factor regulated by autoinhibition

Decreased blood pressure in NOX1-deficient mice

Rho and Rac take center stage

The pathogenesis of atherosclerosis: a perspective for the 1990s

Enhanced superoxide production in experimental venous bypass graft intimal hyperplasia: role of NAD(P)H oxidase.

Regulating actin-filament dynamics in vivo.

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480-487

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10.1161/ATVBAHA.108.181925

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4

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29

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Kathy K. Griendling

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2009-01-15T00:00:00Z

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23797975

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19150879

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2734189

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