Expression of the apoptosis-mediator Fas is enhanced by dysfunctional mitochondria.
about
Caffeic acid phenethyl ester triggers apoptosis through induction of loss of mitochondrial membrane potential in CCRF-CEM cells.Cell death induced by the Fas/Fas ligand pathway and its role in pathology.Geraniin-mediated apoptosis by cleavage of focal adhesion kinase through up-regulation of Fas ligand expression in human melanoma cells.Increased expression but not sensitivity to Fas/CD95 in glioblastoma cells depleted of mitochondrial DNA.Deficiency in a mitochondrial aldehyde dehydrogenase increases vulnerability to oxidative stress in PC12 cells.
P2860
Expression of the apoptosis-mediator Fas is enhanced by dysfunctional mitochondria.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Expression of the apoptosis-mediator Fas is enhanced by dysfunctional mitochondria.
@en
Expression of the apoptosis-mediator Fas is enhanced by dysfunctional mitochondria.
@nl
type
label
Expression of the apoptosis-mediator Fas is enhanced by dysfunctional mitochondria.
@en
Expression of the apoptosis-mediator Fas is enhanced by dysfunctional mitochondria.
@nl
prefLabel
Expression of the apoptosis-mediator Fas is enhanced by dysfunctional mitochondria.
@en
Expression of the apoptosis-mediator Fas is enhanced by dysfunctional mitochondria.
@nl
P2093
P1476
Expression of the apoptosis-mediator Fas is enhanced by dysfunctional mitochondria.
@en
P2093
P304
P356
10.1093/OXFORDJOURNALS.JBCHEM.A021455
P577
1996-09-01T00:00:00Z