During lytic infections, herpes simplex virus type 1 DNA is in complexes with the properties of unstable nucleosomes.
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Role of ND10 nuclear bodies in the chromatin repression of HSV-1A comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivationThe molecular basis of herpes simplex virus latencyThe intrinsic antiviral defense to incoming HSV-1 genomes includes specific DNA repair proteins and is counteracted by the viral protein ICP0.Nuclear IFI16 induction of IRF-3 signaling during herpesviral infection and degradation of IFI16 by the viral ICP0 proteinCellular SNF2H chromatin-remodeling factor promotes herpes simplex virus 1 immediate-early gene expression and replication.The differential mobilization of histones H3.1 and H3.3 by herpes simplex virus 1 relates histone dynamics to the assembly of viral chromatinThe checkpoints of viral gene expression in productive and latent infection: the role of the HDAC/CoREST/LSD1/REST repressor complexGenome wide nucleosome mapping for HSV-1 shows nucleosomes are deposited at preferred positions during lytic infection.Dynamic modulation of HSV chromatin drives initiation of infection and provides targets for epigenetic therapies.An Essential Viral Transcription Activator Modulates Chromatin DynamicsAntiviral activity of PHA767491 against human herpes simplex virus in vitro and in vivo.Visualizing the replicating HSV-1 virus using STED super-resolution microscopy.The dynamics of HCF-1 modulation of herpes simplex virus chromatin during initiation of infection.Cooperative assembly of IFI16 filaments on dsDNA provides insights into host defense strategy.Epigenetic repression of herpes simplex virus infection by the nucleosome remodeler CHD3How to control an infectious bead string: nucleosome-based regulation and targeting of herpesvirus chromatin.Checkpoints in productive and latent infections with herpes simplex virus 1: conceptualization of the issues.Chromatin structure of adenovirus DNA throughout infectionChromatin dynamics during lytic infection with herpes simplex virus 1.Selective recruitment of host factors by HSV-1 replication centers.Core histones H2B and H4 are mobilized during infection with herpes simplex virus 1.Assembly of helper-dependent adenovirus DNA into chromatin promotes efficient gene expression.5-(Perylen-3-yl)ethynyl-arabino-uridine (aUY11), an arabino-based rigid amphipathic fusion inhibitor, targets virion envelope lipids to inhibit fusion of influenza virus, hepatitis C virus, and other enveloped virusesCombinatorial effects of the glucocorticoid receptor and Krüppel-like transcription factor 15 on bovine herpesvirus 1 transcription and productive infection.Chromatin assembly on herpes simplex virus 1 DNA early during a lytic infection is Asf1a dependent.Reversal of heterochromatic silencing of quiescent herpes simplex virus type 1 by ICP0.Herpes simplex virus 1 DNA is in unstable nucleosomes throughout the lytic infection cycle, and the instability of the nucleosomes is independent of DNA replication.Spatiotemporal dynamics of HSV genome nuclear entry and compaction state transitions using bioorthogonal chemistry and super-resolution microscopy.
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P2860
During lytic infections, herpes simplex virus type 1 DNA is in complexes with the properties of unstable nucleosomes.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
During lytic infections, herpe ...... rties of unstable nucleosomes.
@en
During lytic infections, herpe ...... rties of unstable nucleosomes.
@nl
type
label
During lytic infections, herpe ...... rties of unstable nucleosomes.
@en
During lytic infections, herpe ...... rties of unstable nucleosomes.
@nl
prefLabel
During lytic infections, herpe ...... rties of unstable nucleosomes.
@en
During lytic infections, herpe ...... rties of unstable nucleosomes.
@nl
P2860
P356
P1433
P1476
During lytic infections, herpe ...... rties of unstable nucleosomes.
@en
P2093
Jonathan J Lacasse
Luis M Schang
P2860
P304
P356
10.1128/JVI.01934-09
P407
P577
2009-12-09T00:00:00Z