Candida albicans cell surface superoxide dismutases degrade host-derived reactive oxygen species to escape innate immune surveillance.
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SOD Enzymes and Microbial Pathogens: Surviving the Oxidative Storm of InfectionEssential metals at the host-pathogen interface: nutritional immunity and micronutrient assimilation by human fungal pathogensHost-pathogen interactions between the human innate immune system and Candida albicans-understanding and modeling defense and evasion strategiesFungal immune evasion in a model host-pathogen interaction: Candida albicans versus macrophagesHost pathogen relations: exploring animal models for fungal pathogensDevelopment of an in vitro model for the multi-parametric quantification of the cellular interactions between Candida yeasts and phagocytesExtracellular superoxide dismutase protects Histoplasma yeast cells from host-derived oxidative stressCandida albicans SOD5 represents the prototype of an unprecedented class of Cu-only superoxide dismutases required for pathogen defenseAnti-Immune Strategies of Pathogenic FungiCell biology of Candida albicans-host interactionsThe TolC protein of Legionella pneumophila plays a major role in multi-drug resistance and the early steps of host invasionCandida albicans suppresses nitric oxide generation from macrophages via a secreted moleculeA Multifaceted Study of Scedosporium boydii Cell Wall Changes during Germination and Identification of GPI-Anchored ProteinsThe Candida albicans Histone Acetyltransferase Hat1 Regulates Stress Resistance and Virulence via Distinct Chromatin Assembly PathwaysJagunal homolog 1 is a critical regulator of neutrophil function in fungal host defenseLive imaging of disseminated candidiasis in zebrafish reveals role of phagocyte oxidase in limiting filamentous growth.Pho4 mediates phosphate acquisition in Candida albicans and is vital for stress resistance and metal homeostasisA 5' UTR-mediated translational efficiency mechanism inhibits the Candida albicans morphological transition.Elevated catalase expression in a fungal pathogen is a double-edged sword of iron.Bridging the Gap to Non-toxic Fungal Control: Lupinus-Derived Blad-Containing Oligomer as a Novel Candidate to Combat Human Pathogenic Fungi.Additive contributions of two manganese-cored superoxide dismutases (MnSODs) to antioxidation, UV tolerance and virulence of Beauveria bassianaIdentification of GIG1, a GlcNAc-induced gene in Candida albicans needed for normal sensitivity to the chitin synthase inhibitor nikkomycin Z.A histone deacetylase adjusts transcription kinetics at coding sequences during Candida albicans morphogenesis.Cellular responses of Candida albicans to phagocytosis and the extracellular activities of neutrophils are critical to counteract carbohydrate starvation, oxidative and nitrosative stressNews from the fungal front: wall proteome dynamics and host-pathogen interplayInterspecies protein-protein interaction network construction for characterization of host-pathogen interactions: a Candida albicans-zebrafish interaction study.Novel insight into neutrophil immune responses by dry mass determination of Candida albicans morphotypes.Repurposing as a means to increase the activity of amphotericin B and caspofungin against Candida albicans biofilms.Oxidative stress responses in the human fungal pathogen, Candida albicans.Systemic oxidative stress could predict assisted reproductive technique outcomeAntibacterial and Antifungal Activity of ZnO Containing GlassesCsr1/Zap1 Maintains Zinc Homeostasis and Influences Virulence in Candida dubliniensis but Is Not Coupled to Morphogenesis.Identification and Analysis of the Role of Superoxide Dismutases Isoforms in the Pathogenesis of Paracoccidioides spp.Candida albicans adapts to host copper during infection by swapping metal cofactors for superoxide dismutaseAn Adaptation to Low Copper in Candida albicans Involving SOD Enzymes and the Alternative Oxidase.The Iron-Dependent Regulation of the Candida albicans Oxidative Stress Response by the CCAAT-Binding Factor.Normal adaptation of Candida albicans to the murine gastrointestinal tract requires Efg1p-dependent regulation of metabolic and host defense genes.Cryptococcus neoformans phosphoinositide-dependent kinase 1 (PDK1) ortholog is required for stress tolerance and survival in murine phagocytesThe role of Candida albicans AP-1 protein against host derived ROS in in vivo models of infection.Surface stress induces a conserved cell wall stress response in the pathogenic fungus Candida albicans.
P2860
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P2860
Candida albicans cell surface superoxide dismutases degrade host-derived reactive oxygen species to escape innate immune surveillance.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Candida albicans cell surface ...... pe innate immune surveillance.
@en
Candida albicans cell surface ...... pe innate immune surveillance.
@nl
type
label
Candida albicans cell surface ...... pe innate immune surveillance.
@en
Candida albicans cell surface ...... pe innate immune surveillance.
@nl
prefLabel
Candida albicans cell surface ...... pe innate immune surveillance.
@en
Candida albicans cell surface ...... pe innate immune surveillance.
@nl
P2093
P2860
P1476
Candida albicans cell surface ...... ape innate immune surveillance
@en
P2093
Christelle Bourgeois
Ingrid E Frohner
Kristina Yatsyk
Olivia Majer
P2860
P304
P356
10.1111/J.1365-2958.2008.06528.X
P407
P50
P577
2008-11-04T00:00:00Z