O-GLcNAc post-translational modifications regulate the entry of neurons into an axon branching program.
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Cytosine modifications in neurodevelopment and diseasesThree Decades of Research on O-GlcNAcylation - A Major Nutrient Sensor That Regulates Signaling, Transcription and Cellular MetabolismUnlocking epigenetic codes in neurogenesisFunctional O-GlcNAc modifications: implications in molecular regulation and pathophysiologyThe cytoskeletal and signaling mechanisms of axon collateral branchingDevelopmental regulation of protein O-GlcNAcylation, O-GlcNAc transferase, and O-GlcNAcase in mammalian brainGlobal identification and characterization of both O-GlcNAcylation and phosphorylation at the murine synapseO-GlcNAcase: promiscuous hexosaminidase or key regulator of O-GlcNAc signaling?O-linked beta-N-acetylglucosamine (O-GlcNAc): Extensive crosstalk with phosphorylation to regulate signaling and transcription in response to nutrients and stressRegulation of Dauer formation by O-GlcNAcylation in Caenorhabditis elegans.The emerging link between O-GlcNAc and Alzheimer disease.Dynamic O-GlcNAc modification regulates CREB-mediated gene expression and memory formationO-GlcNAcomics--Revealing roles of O-GlcNAcylation in disease mechanisms and development of potential diagnostics.Chemical Modulation of Protein O-GlcNAcylation via OGT Inhibition Promotes Human Neural Cell Differentiation.Differential effects of PKA-controlled CaMKK2 variants on neuronal differentiationO-GlcNAc Transferase Is Essential for Sensory Neuron Survival and Maintenance.Angioneural crosstalk in scaffolds with oriented microchannels for regenerative spinal cord injury repair.Synaptotagmin-1 promotes the formation of axonal filopodia and branches along the developing axons of forebrain neurons.Nutrient-driven O-linked N-acetylglucosamine (O-GlcNAc) cycling impacts neurodevelopmental timing and metabolism.The O-GlcNAc Modification of CDK5 Involved in Neuronal Apoptosis Following In Vitro Intracerebral Hemorrhage.The O-GlcNAc Transferase Intellectual Disability Mutation L254F Distorts the TPR Helix.O-GlcNAc cycling in the developing, adult and geriatric brain.'-GlcNAc Code' Mediated Biological Functions of Downstream Proteins
P2860
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P2860
O-GLcNAc post-translational modifications regulate the entry of neurons into an axon branching program.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
O-GLcNAc post-translational mo ...... nto an axon branching program.
@en
O-GLcNAc post-translational mo ...... nto an axon branching program.
@nl
type
label
O-GLcNAc post-translational mo ...... nto an axon branching program.
@en
O-GLcNAc post-translational mo ...... nto an axon branching program.
@nl
prefLabel
O-GLcNAc post-translational mo ...... nto an axon branching program.
@en
O-GLcNAc post-translational mo ...... nto an axon branching program.
@nl
P2093
P2860
P356
P1476
O-GLcNAc post-translational mo ...... nto an axon branching program.
@en
P2093
Gianluca Gallo
Herb Francisco
Katherine Kollins
Keith Vosseller
Neal Varghis
P2860
P304
P356
10.1002/DNEU.20695
P577
2009-02-01T00:00:00Z