COX2 inhibitors for acquired brain injuries: is the time ripe? - Wikidata - awgldk - TriplyDB

COX2 inhibitors for acquired brain injuries: is the time ripe?

COX2 inhibitors for acquired brain injuries: is the time ripe?

http://www.wikidata.org/entity/Q42564198

about

Neuronal and nonneuronal COX-2 expression confers neurotoxic and neuroprotective phenotypes in response to excitotoxin challenge Strategies to defeat ketamine-induced neonatal brain injury Using anesthetics and analgesics in experimental traumatic brain injury

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COX2 inhibitors for acquired brain injuries: is the time ripe?

http://www.wikidata.org/entity/Q42564198

description

2010 nî lūn-bûn

@nan

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

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2010年論文

@zh-tw

2010年论文

@wuu

2010年论文

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2010年论文

@zh-cn

name

COX2 inhibitors for acquired brain injuries: is the time ripe?

@en

COX2 inhibitors for acquired brain injuries: is the time ripe?

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type

label

COX2 inhibitors for acquired brain injuries: is the time ripe?

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COX2 inhibitors for acquired brain injuries: is the time ripe?

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prefLabel

COX2 inhibitors for acquired brain injuries: is the time ripe?

@en

COX2 inhibitors for acquired brain injuries: is the time ripe?

@nl

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Critical Care Medicine

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COX2 inhibitors for acquired brain injuries: is the time ripe?

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Kenneth I Strauss

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P2860

Risk of myocardial infarction in patients taking cyclo-oxygenase-2 inhibitors or conventional non-steroidal anti-inflammatory drugs: population based nested case-control analysis

Cardiovascular events associated with rofecoxib in a colorectal adenoma chemoprevention trial

Anti-inflammatory properties of cytochrome P450 epoxygenase-derived eicosanoids.

Cyclooxygenase-2 inhibition provides lasting protection against neonatal hypoxic-ischemic brain injury.

Complications of the COX-2 inhibitors parecoxib and valdecoxib after cardiac surgery.

Cyclooxygenase-2-specific inhibitor improves functional outcomes, provides neuroprotection, and reduces inflammation in a rat model of traumatic brain injury.

Cyclooxygenase-2 (COX-2) in inflammatory and degenerative brain diseases.

Antiinflammatory and neuroprotective actions of COX2 inhibitors in the injured brain.

12-hydroxyeicosatetrenoate (12-HETE) attenuates AMPA receptor-mediated neurotoxicity: evidence for a G-protein-coupled HETE receptor.

The highly selective cyclooxygenase-2 inhibitor DFU is neuroprotective when given several hours after transient cerebral ischemia in gerbils.

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