Multiple pathways to long-lasting phrenic motor facilitation. - Wikidata - awgldk - TriplyDB

Multiple pathways to long-lasting phrenic motor facilitation.

Multiple pathways to long-lasting phrenic motor facilitation.

http://www.wikidata.org/entity/Q42573464

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Hypoxia-induced phrenic long-term facilitation: emergent properties Exercise, neurotrophins, and axon regeneration in the PNS Unexpected benefits of intermittent hypoxia: enhanced respiratory and nonrespiratory motor function Neither serotonin nor adenosine-dependent mechanisms preserve ventilatory capacity in ALS rats Adrenergic α₁ receptor activation is sufficient, but not necessary for phrenic long-term facilitation.Repetitive intermittent hypoxia induces respiratory and somatic motor recovery after chronic cervical spinal injury Adenosine 2A receptor inhibition enhances intermittent hypoxia-induced diaphragm but not intercostal long-term facilitation.Lipopolysaccharide attenuates phrenic long-term facilitation following acute intermittent hypoxia.Diaphragm long-term facilitation following acute intermittent hypoxia during wakefulness and sleep.Therapeutic potential of intermittent hypoxia: a matter of dose.Spinal nNOS regulates phrenic motor facilitation by a 5-HT2B receptor- and NADPH oxidase-dependent mechanism.Spinal vascular endothelial growth factor induces phrenic motor facilitation via extracellular signal-regulated kinase and Akt signaling.Repetitive acute intermittent hypoxia increases expression of proteins associated with plasticity in the phrenic motor nucleus Similarities and differences in mechanisms of phrenic and hypoglossal motor facilitation.Phrenic long-term facilitation requires PKCθ activity within phrenic motor neurons.Acute intermittent hypoxia-induced expression of brain-derived neurotrophic factor is disrupted in the brainstem of methyl-CpG-binding protein 2 null mice.The effect of tongue exercise on serotonergic input to the hypoglossal nucleus in young and old rats.Severe acute intermittent hypoxia elicits phrenic long-term facilitation by a novel adenosine-dependent mechanism.Mammalian target of rapamycin is required for phrenic long-term facilitation following severe but not moderate acute intermittent hypoxia.Spinal 5-HT7 receptors induce phrenic motor facilitation via EPAC-mTORC1 signaling.Acute intermittent hypoxia induced phrenic long-term facilitation despite increased SOD1 expression in a rat model of ALS.Phrenic long-term facilitation after acute intermittent hypoxia requires spinal ERK activation but not TrkB synthesis Intermittent hypoxia and neurorehabilitation.Cervical spinal erythropoietin induces phrenic motor facilitation via extracellular signal-regulated protein kinase and Akt signaling.Daily acute intermittent hypoxia elicits functional recovery of diaphragm and inspiratory intercostal muscle activity after acute cervical spinal injury Spinal atypical protein kinase C activity is necessary to stabilize inactivity-induced phrenic motor facilitation.Inactivity-induced phrenic and hypoglossal motor facilitation are differentially expressed following intermittent vs. sustained neural apnea.Ventilatory long-term facilitation is evident after initial and repeated exposure to intermittent hypoxia in mice genetically depleted of brain serotonin Time Domains of the Hypoxic Ventilatory Response and Their Molecular Basis.Sustained Hypoxia Elicits Competing Spinal Mechanisms of Phrenic Motor Facilitation.Intermittent hypoxia, respiratory plasticity and sleep apnea in humans: present knowledge and future investigations.Changes in neurochemicals within the ventrolateral medullary respiratory column in awake goats after carotid body denervation.Spinal 5-HT7 receptors and protein kinase A constrain intermittent hypoxia-induced phrenic long-term facilitation.Repeated intravenous doxapram induces phrenic motor facilitation.Inactivity-induced respiratory plasticity: protecting the drive to breathe in disorders that reduce respiratory neural activity Enhanced recovery of breathing capacity from combined adenosine 2A receptor inhibition and daily acute intermittent hypoxia after chronic cervical spinal injury.Repetitive acute intermittent hypoxia increases growth/neurotrophic factor expression in non-respiratory motor neurons Spinal plasticity following intermittent hypoxia: implications for spinal injury.Treatments to restore respiratory function after spinal cord injury and their implications for regeneration, plasticity and adaptation.Common mechanisms of compensatory respiratory plasticity in spinal neurological disorders.

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P2860

Multiple pathways to long-lasting phrenic motor facilitation.

http://www.wikidata.org/entity/Q42573464

description

2010 nî lūn-bûn

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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2010年论文

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2010年论文

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name

Multiple pathways to long-lasting phrenic motor facilitation.

@en

Multiple pathways to long-lasting phrenic motor facilitation.

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type

label

Multiple pathways to long-lasting phrenic motor facilitation.

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Multiple pathways to long-lasting phrenic motor facilitation.

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Multiple pathways to long-lasting phrenic motor facilitation.

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Multiple pathways to long-lasting phrenic motor facilitation.

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978-1-4419-5692-7_45

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Advances in Experimental Medicine and Biology

P1476

Multiple pathways to long-lasting phrenic motor facilitation.

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P2093

Erica A Dale-Nagle

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Gordon S Mitchell

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Michael S Hoffman

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Peter M MacFarlane

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P2860

BDNF is necessary and sufficient for spinal respiratory plasticity following intermittent hypoxia

Phrenic long-term facilitation requires 5-HT receptor activation during but not following episodic hypoxia.

Long term facilitation of phrenic motor output.

Daily intermittent hypoxia augments spinal BDNF levels, ERK phosphorylation and respiratory long-term facilitation.

Neuroplasticity in respiratory motor control.

Neuronal 5-HT metabotropic receptors: fine-tuning of their structure, signaling, and roles in synaptic modulation.

Is there a link between intermittent hypoxia-induced respiratory plasticity and obstructive sleep apnoea?

Formation and maintenance of ventilatory long-term facilitation require NMDA but not non-NMDA receptors in awake rats.

Respiratory plasticity following intermittent hypoxia: roles of protein phosphatases and reactive oxygen species.

Reactive oxygen species and respiratory plasticity following intermittent hypoxia.

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225-230

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10.1007/978-1-4419-5692-7_45

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669

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41850315

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20217354

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