Pneumocystis cell wall beta-glucans stimulate alveolar epithelial cell chemokine generation through nuclear factor-kappaB-dependent mechanisms. - Wikidata - awgldk - TriplyDB

Pneumocystis cell wall beta-glucans stimulate alveolar epithelial cell chemokine generation through nuclear factor-kappaB-dependent mechanisms.

Pneumocystis cell wall beta-glucans stimulate alveolar epithelial cell chemokine generation through nuclear factor-kappaB-dependent mechanisms.

http://www.wikidata.org/entity/Q42575251

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Characterization of a distinct host response profile to Pneumocystis murina asci during clearance of pneumocystis pneumonia The use of lentinan for treating gastric cancer Pathobiology of Pneumocystis pneumonia: life cycle, cell wall and cell signal transduction Can host receptors for fungi be targeted for treatment of fungal infections?Echinocandin treatment of pneumocystis pneumonia in rodent models depletes cysts leaving trophic burdens that cannot transmit the infection Inhaled innate immune ligands to prevent pneumonia.Serum markers in interstitial pneumonia with and without Pneumocystis jirovecii colonization: a prospective study Stimulated innate resistance of lung epithelium protects mice broadly against bacteria and fungi.Immune modulation with sulfasalazine attenuates immunopathogenesis but enhances macrophage-mediated fungal clearance during Pneumocystis pneumonia.Pneumocystis cell wall beta-glucan stimulates calcium-dependent signaling of IL-8 secretion by human airway epithelial cells.Host lung gene expression patterns predict infectious etiology in a mouse model of pneumonia.Toll-like receptor 2 mediates alveolar macrophage response to Pneumocystis murina IL-33 and M2a alveolar macrophages promote lung defense against the atypical fungal pathogen Pneumocystis murina.Fungal allergen β-glucans trigger p38 mitogen-activated protein kinase-mediated IL-6 translation in lung epithelial cells Evidence for Proinflammatory β-1,6 Glucans in the Pneumocystis carinii Cell Wall.Inducible innate resistance of lung epithelium to infection.Dectin-1 is required for beta-glucan recognition and control of fungal infection.Epithelial cells and innate antifungal defense.Inhaled therapeutics for prevention and treatment of pneumonia Scavenger receptor A dampens induction of inflammation in response to the fungal pathogen Pneumocystis carinii.Characterization of chemokine and chemokine receptor expression during Pneumocystis infection in healthy and immunodeficient mice.Glycosphingolipids mediate pneumocystis cell wall β-glucan activation of the IL-23/IL-17 axis in human dendritic cells.Alveolar macrophages in neonatal mice are inherently unresponsive to Pneumocystis murina infection.Regulation of interleukin-6 secretion by the two-pore-domain potassium channel Trek-1 in alveolar epithelial cells.Src kinase-targeted anti-inflammatory activity of davallialactone from Inonotus xeranticus in lipopolysaccharide-activated RAW264.7 cells.Beyond inflammation: airway epithelial cells are at the interface of innate and adaptive immunity Current understanding of Pneumocystis immunology.The biological role of dectin-1 in immune response.Biofilm formation by Pneumocystis spp Parenchymal cell TNF receptors contribute to inflammatory cell recruitment and respiratory failure in Pneumocystis carinii-induced pneumonia.The absence of Hck, Fgr, and Lyn tyrosine kinases augments lung innate immune responses to Pneumocystis murina.B cell production of tumor necrosis factor in response to Pneumocystis murina infection in mice.Effects of decreased calmodulin protein on the survival mechanisms of alveolar macrophages during Pneumocystis pneumonia Beta-glucans as immunostimulant in vertebrates and invertebrates.Chitinases in Pneumocystis carinii pneumonia Inhibitor κBα protein therapy alleviates severe pneumonia through inhibition of nuclear factor κB Innate immunity in the respiratory epithelium.Natural glycans and glycoconjugates as immunomodulating agents.Immune Modulation as Adjunctive Therapy for Pneumocystis pneumonia.The Interaction of Pneumocystis with the C-Type Lectin Receptor Mincle Exerts a Significant Role in Host Defense against Infection.

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Pneumocystis cell wall beta-glucans stimulate alveolar epithelial cell chemokine generation through nuclear factor-kappaB-dependent mechanisms.

http://www.wikidata.org/entity/Q42575251

description

2005 nî lūn-bûn

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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2005年论文

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name

Pneumocystis cell wall beta-gl ...... r-kappaB-dependent mechanisms.

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Pneumocystis cell wall beta-gl ...... r-kappaB-dependent mechanisms.

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type

label

Pneumocystis cell wall beta-gl ...... r-kappaB-dependent mechanisms.

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Pneumocystis cell wall beta-gl ...... r-kappaB-dependent mechanisms.

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Pneumocystis cell wall beta-gl ...... r-kappaB-dependent mechanisms.

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Pneumocystis cell wall beta-gl ...... r-kappaB-dependent mechanisms.

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RCMB.2004-0300OC

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American Journal of Respiratory Cell and Molecular Biology

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Pneumocystis cell wall beta-gl ...... r-kappaB-dependent mechanisms.

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P2093

Andrew H Limper

http://www.w3.org/2001/XMLSchema#string

Frances McCann

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Peter Y Hahn

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Scott E Evans

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Theodore J Kottom

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Zvezdana Vuk Pavlovic'

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P2860

Deregulation of NF-kappaB and its upstream kinases in cancer

Complex formation and cooperation of protein kinase C theta and Akt1/protein kinase B alpha in the NF-kappa B transactivation cascade in Jurkat T cells

The beta-glucan receptor, dectin-1, is predominantly expressed on the surface of cells of the monocyte/macrophage and neutrophil lineages

Crocidolite activates NF-kappa B and MIP-2 gene expression in rat alveolar epithelial cells. Role of mitochondrial-derived oxidants

Dectin-1 is a major beta-glucan receptor on macrophages

MAPK-activated protein kinase-2 participates in p38 MAPK-dependent and ERK-dependent functions in human neutrophils

Regulation of chemokine mRNA expression in a rat model of vanadium-induced pulmonary inflammation.

Alveolar macrophage interactions with Pneumocystis carinii.

Chemokine gene expression during Pneumocystis carinii-driven pulmonary inflammation.

Immune-mediated inflammation directly impairs pulmonary function, contributing to the pathogenesis of Pneumocystis carinii pneumonia

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490-497

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10.1165/RCMB.2004-0300OC

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6

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32

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15746433

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2715319

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