Injury-induced decline of intrinsic regenerative ability revealed by quantitative proteomics. - Wikidata - awgldk - TriplyDB

Injury-induced decline of intrinsic regenerative ability revealed by quantitative proteomics.

Injury-induced decline of intrinsic regenerative ability revealed by quantitative proteomics.

http://www.wikidata.org/entity/Q42579331

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What makes a RAG regeneration associated?Emergent properties of neural repair: elemental biology to therapeutic concepts Intrinsic Axonal Growth and the Drive for Regeneration Expressing Constitutively Active Rheb in Adult Neurons after a Complete Spinal Cord Injury Enhances Axonal Regeneration beyond a Chondroitinase-Treated Glial Scar.The Potential of Human Stem Cells for the Study and Treatment of Glaucoma.Signaling Over Distances.Molecular and Cellular Mechanisms of Axonal Regeneration After Spinal Cord Injury.Reassembly of Excitable Domains after CNS Axon Regeneration.Epigenetic profiling reveals a developmental decrease in promoter accessibility during cortical maturation in vivo.Rapamycin-Resistant mTOR Activity Is Required for Sensory Axon Regeneration Induced by a Conditioning Lesion.Bone Marrow-Derived Cells as a Therapeutic Approach to Optic Nerve Diseases.The application of CRISPR technology to high content screening in primary neurons.Non-coding RNAs as Emerging Regulators of Neural Injury Responses and Regeneration.CNS repair and axon regeneration: Using genetic variation to determine mechanisms.The age factor in axonal repair after spinal cord injury: A focus on neuron-intrinsic mechanisms.Selecting optimal combinations of transcription factors to promote axon regeneration: Why mechanisms matter.Optic nerve regeneration in mammals: Regenerated or spared axons?Gene Manipulation Strategies to Identify Molecular Regulators of Axon Regeneration in the Central Nervous System.The tumor suppressor HHEX inhibits axon growth when prematurely expressed in developing central nervous system neurons.RhoA knockdown by cationic amphiphilic copolymer/siRhoA polyplexes enhances axonal regeneration in rat spinal cord injury model.Damage-induced neuronal endopeptidase (DINE) enhances axonal regeneration potential of retinal ganglion cells after optic nerve injury.Restoration of Visual Function by Enhancing Conduction in Regenerated Axons Targeting 14-3-3 adaptor protein-protein interactions to stimulate central nervous system repair.3D Visualization of Individual Regenerating Retinal Ganglion Cell Axons Reveals Surprisingly Complex Growth Paths.NT-3 promotes proprioceptive axon regeneration when combined with activation of the mTor intrinsic growth pathway but not with reduction of myelin extrinsic inhibitors.Boosting Central Nervous System Axon Regeneration by Circumventing Limitations of Natural Cytokine Signaling.A high mitochondrial transport rate characterizes CNS neurons with high axonal regeneration capacity.Two PTP receptors mediate CSPG inhibition by convergent and divergent signaling pathways in neurons.Neuroproteomics: How Many Angels can be Identified in an Extract from the Head of a Pin?Zinc chelation and Klf9 knockdown cooperatively promote axon regeneration after optic nerve injury.The Mammalian-Specific Protein Armcx1 Regulates Mitochondrial Transport during Axon Regeneration.Labeling and identifying cell-specific proteomes in the mouse brain.Complementary research models and methods to study axonal regeneration in the vertebrate retinofugal system.Maximizing functional axon repair in the injured central nervous system: Lessons from neuronal development.Can injured adult CNS axons regenerate by recapitulating development?Defining the temporal course of murine neurofibromatosis-1 optic gliomagenesis reveals a therapeutic window to attenuate retinal dysfunction.Optic nerve regeneration in the mouse is a complex trait modulated by genetic background.Epitranscriptomic m6A Regulation of Axon Regeneration in the Adult Mammalian Nervous System.An Intrinsic Epigenetic Barrier for Functional Axon Regeneration.Intrinsic mechanisms of neuronal axon regeneration.

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Injury-induced decline of intrinsic regenerative ability revealed by quantitative proteomics.

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Injury-induced decline of intr ...... ed by quantitative proteomics.

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Injury-induced decline of intr ...... ed by quantitative proteomics.

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Injury-induced decline of intr ...... ed by quantitative proteomics.

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Alban Latremoliere

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Ceren Uncu

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Chen Wang

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Clifford J Woolf

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Judith A Steen

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Peter Warren

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Shaojun Tang

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Zhigang He

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P2860

PTEN deletion enhances the regenerative ability of adult corticospinal neurons

mTOR signaling in growth control and disease

Promoting axon regeneration in the adult CNS by modulation of the PTEN/mTOR pathway

Replicate high-density rat genome oligonucleotide microarrays reveal hundreds of regulated genes in the dorsal root ganglion after peripheral nerve injury

Systematic and integrative analysis of large gene lists using DAVID bioinformatics resources

Fibroblast growth factor-inducible-14 is induced in axotomized neurons and promotes neurite outgrowth

Defining the role of mTOR in cancer

c-Myc is a universal amplifier of expressed genes in lymphocytes and embryonic stem cells

Protein phosphatase 2A facilitates axonogenesis by dephosphorylating CRMP2

The insulin-like growth factor 1 receptor is essential for axonal regeneration in adult central nervous system neurons

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