Arterial eicosanoid production following chronic exposure to a periarterial haematoma.
about
The importance of early brain injury after subarachnoid hemorrhageOral nimodipine reduces prostaglandin and thromboxane production by arteries chronically exposed to a periarterial haematoma and the antifibrinolytic agent tranexamic acid.In vivo animal models of cerebral vasospasm: a review.Mechanisms of acute brain injury after subarachnoid hemorrhage.Aneurysmal subarachnoid hemorrhage models: do they need a fix?Does thrombin prevent cerebral vasospasm following aneurysmal subarachnoid haemorrhage?Spectrum of altered reactivity of isolated cerebral arteries following subarachnoid haemorrhage--response to potassium, pH, noradrenaline, 5-hydroxytryptamine, and sodium loading.
P2860
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P2860
Arterial eicosanoid production following chronic exposure to a periarterial haematoma.
description
1984 nî lūn-bûn
@nan
1984年の論文
@ja
1984年論文
@yue
1984年論文
@zh-hant
1984年論文
@zh-hk
1984年論文
@zh-mo
1984年論文
@zh-tw
1984年论文
@wuu
1984年论文
@zh
1984年论文
@zh-cn
name
Arterial eicosanoid production following chronic exposure to a periarterial haematoma.
@en
Arterial eicosanoid production following chronic exposure to a periarterial haematoma.
@nl
type
label
Arterial eicosanoid production following chronic exposure to a periarterial haematoma.
@en
Arterial eicosanoid production following chronic exposure to a periarterial haematoma.
@nl
prefLabel
Arterial eicosanoid production following chronic exposure to a periarterial haematoma.
@en
Arterial eicosanoid production following chronic exposure to a periarterial haematoma.
@nl
P2093
P2860
P356
P1476
Arterial eicosanoid production following chronic exposure to a periarterial haematoma.
@en
P2093
P2860
P304
P356
10.1136/JNNP.47.7.661
P407
P577
1984-07-01T00:00:00Z