Alterations in excitation-contraction coupling in chronically ischemic or hibernating myocardium

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New vessel formation in the context of cardiomyocyte regeneration--the role and importance of an adequate perfusing vasculature.Metabolic inhibition reduces cardiac L-type Ca2+ channel current due to acidification caused by ATP hydrolysis

P2860

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P2860

Alterations in excitation-contraction coupling in chronically ischemic or hibernating myocardium

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http://www.wikidata.org/entity/Q42584078

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2005 nî lūn-bûn

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2005年の論文

@ja

2005年論文

@yue

2005年論文

@zh-hant

2005年論文

@zh-hk

2005年論文

@zh-mo

2005年論文

@zh-tw

2005年论文

@wuu

2005年论文

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2005年论文

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name

Alterations in excitation-cont ...... emic or hibernating myocardium

@en

Alterations in excitation-cont ...... emic or hibernating myocardium

@nl

type

Item

label

Alterations in excitation-cont ...... emic or hibernating myocardium

@en

Alterations in excitation-cont ...... emic or hibernating myocardium

@nl

prefLabel

Alterations in excitation-cont ...... emic or hibernating myocardium

@en

Alterations in excitation-cont ...... emic or hibernating myocardium

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P1476

Alterations in excitation-cont ...... emic or hibernating myocardium

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P2093

Bart Bijnens

http://www.w3.org/2001/XMLSchema#string

Erik Verbeken

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Frank R Heinzel

http://www.w3.org/2001/XMLSchema#string

Ger Stienen

http://www.w3.org/2001/XMLSchema#string

Karin R Sipido

http://www.w3.org/2001/XMLSchema#string

Piet Claus

http://www.w3.org/2001/XMLSchema#string

Virginie Bito

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P2860

Force production in mechanically isolated cardiac myocytes from human ventricular muscle tissue.

Resting myocardial flow in hibernating myocardium: validating animal models of human pathophysiology.

New therapeutic options in congestive heart failure: Part I.

New therapeutic options in congestive heart failure: Part II.

Myocardial blood flow in patients with hibernating myocardium.

A perspective on the three large multicenter randomized clinical trials of coronary bypass surgery for chronic stable angina.

Chronic myocardial hibernation in humans. From bedside to bench.

Pathophysiological mechanisms of chronic reversible left ventricular dysfunction due to coronary artery disease (hibernating myocardium).

Protein kinase A hyperphosphorylation increases basal current but decreases beta-adrenergic responsiveness of the sarcolemmal Na+-Ca2+ exchanger in failing pig myocytes.

Progressive loss of perfusion-contraction matching during sustained moderate ischemia in pigs.

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scholarly article

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Jolanda van der Velden

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2005-01-01T00:00:00Z

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19641678

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P921

hibernation

P932

2716242

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Alterations in excitation-contraction coupling in chronically ischemic or hibernating myocardium

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P2860

P2860

Alterations in excitation-contraction coupling in chronically ischemic or hibernating myocardium

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P1433

P1476

P2093

P2860

P304

P31

P433

P478

P50

P577

P698

P921

P932

P1433

P1476

P2093

P2860

P304

P31

P433

P478

P50

P577

P698

P921

P932