A RNA interference screen identifies RIP3 as an essential inducer of TNF-induced programmed necrosis.

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Programmed necrosis in the cross talk of cell death and inflammation RIP1-dependent and independent effects of necrostatin-1 in necrosis and T cell activation Autophagy, apoptosis, mitoptosis and necrosis: interdependence between those pathways and effects on cancer.

P2860

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A RNA interference screen identifies RIP3 as an essential inducer of TNF-induced programmed necrosis.

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http://www.wikidata.org/entity/Q42585640

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2011 nî lūn-bûn

@nan

2011年の論文

@ja

2011年論文

@yue

2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

2011年论文

@zh

2011年论文

@zh-cn

name

A RNA interference screen iden ...... F-induced programmed necrosis.

@en

A RNA interference screen iden ...... F-induced programmed necrosis.

@nl

type

Item

label

A RNA interference screen iden ...... F-induced programmed necrosis.

@en

A RNA interference screen iden ...... F-induced programmed necrosis.

@nl

prefLabel

A RNA interference screen iden ...... F-induced programmed necrosis.

@en

A RNA interference screen iden ...... F-induced programmed necrosis.

@nl

P1476

A RNA interference screen iden ...... F-induced programmed necrosis.

@en

P2093

Sreerupa Challa

http://www.w3.org/2001/XMLSchema#string

Youngsik Cho

http://www.w3.org/2001/XMLSchema#string

P2860

Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule

Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes

Riboflavin kinase couples TNF receptor 1 to NADPH oxidase

Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha

Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation

FADD and caspase-8 control the outcome of autophagic signaling in proliferating T cells

Pleiotropic defects in lymphocyte activation caused by caspase-8 mutations lead to human immunodeficiency

Fas-mediated apoptosis and activation-induced T-cell proliferation are defective in mice lacking FADD/Mort1

RIP3, an energy metabolism regulator that switches TNF-induced cell death from apoptosis to necrosis

How dying cells alert the immune system to danger

P304

589-593

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P31

scholarly article

P356

10.1007/978-1-4419-6612-4_62

http://www.w3.org/2001/XMLSchema#string

P407

English

P478

691

http://www.w3.org/2001/XMLSchema#string

P50

Francis Ka-Ming Chan

P577

2011-01-01T00:00:00Z

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P698

21153365

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P932

3763504

http://www.w3.org/2001/XMLSchema#string

A RNA interference screen identifies RIP3 as an essential inducer of TNF-induced programmed necrosis.

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P2860

P2860

A RNA interference screen identifies RIP3 as an essential inducer of TNF-induced programmed necrosis.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P478

P50

P577

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P478

P50

P577

P698

P932