NADPH oxidase 4 mediates TGF-β-induced smooth muscle α-actin via p38MAPK and serum response factor - Wikidata - awgldk - TriplyDB

NADPH oxidase 4 mediates TGF-β-induced smooth muscle α-actin via p38MAPK and serum response factor

NADPH oxidase 4 mediates TGF-β-induced smooth muscle α-actin via p38MAPK and serum response factor

http://www.wikidata.org/entity/Q42588680

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Suppression of NADPH Oxidase Activity May Slow the Expansion of Osteolytic Bone Metastases The role of AST-120 and protein-bound uremic toxins in irritable bowel syndrome: a therapeutic perspective Biochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular system Extracellular matrix as a contextual determinant of transforming growth factor-β signaling in epithelial-mesenchymal transition and in cancer Redox signaling in cardiovascular pathophysiology: A focus on hydrogen peroxide and vascular smooth muscle cells Regulation of signal transduction by reactive oxygen species in the cardiovascular system Oxidases and peroxidases in cardiovascular and lung disease: new concepts in reactive oxygen species signaling Decoding NADPH oxidase 4 expression in human tumors.Negative regulation of NADPH oxidase 4 by hydrogen peroxide-inducible clone 5 (Hic-5) protein Expression of NADPH oxidase (NOX) 5 in rabbit corneal stromal cells.NADPH oxidase NOX4 mediates stellate cell activation and hepatocyte cell death during liver fibrosis development.Transforming growth factor β inhibits platelet derived growth factor-induced vascular smooth muscle cell proliferation via Akt-independent, Smad-mediated cyclin D1 downregulation Transforming growth factor-beta1 (TGF-beta1) utilizes distinct pathways for the transcriptional activation of microRNA 143/145 in human coronary artery smooth muscle cells.Hic-5 Mediates TGFβ-Induced Adhesion in Vascular Smooth Muscle Cells by a Nox4-Dependent Mechanism.Galangin attenuates airway remodelling by inhibiting TGF-β1-mediated ROS generation and MAPK/Akt phosphorylation in asthma Transforming growth factor-β1 requires NADPH oxidase 4 for angiogenesis in vitro and in vivo Redox-Sensitive Regulation of Myocardin-Related Transcription Factor (MRTF-A) Phosphorylation via Palladin in Vascular Smooth Muscle Cell Differentiation Marker Gene Expression The NOX toolbox: validating the role of NADPH oxidases in physiology and disease.A TRPC6-dependent pathway for myofibroblast transdifferentiation and wound healing in vivo.The role of Nox-mediated oxidation in the regulation of cytoskeletal dynamics NADPH oxidase 4 is required for interleukin-1β-mediated activation of protein kinase Cδ and downstream activation of c-jun N-terminal kinase signaling in smooth muscle.Fuzheng Huayu Recipe Ameliorates Liver Fibrosis by Restoring Balance between Epithelial-to-Mesenchymal Transition and Mesenchymal-to-Epithelial Transition in Hepatic Stellate Cells.Reactive oxygen species in vascular formation and development.miR-9-5p suppresses pro-fibrogenic transformation of fibroblasts and prevents organ fibrosis by targeting NOX4 and TGFBR2 Lung extracellular matrix and redox regulation Inhibiting TGFβ1 has a protective effect on mouse bone marrow suppression following ionizing radiation exposure in vitro Nox4 Plays a Role in TGF-β-Dependent Lens Epithelial to Mesenchymal Transition NADPH oxidase-dependent redox signaling in TGF-β-mediated fibrotic responses.Signaling mechanisms that regulate smooth muscle cell differentiation.Differential roles of NADPH oxidases in vascular physiology and pathophysiology Oxidative stress, Nox isoforms and complications of diabetes--potential targets for novel therapies.Smooth Muscle Cell Differentiation: Model Systems, Regulatory Mechanisms, and Vascular Diseases.Redox regulation of vascular remodeling.CVD and Oxidative Stress.Role of TGF-β1/p38 MAPK pathway in hepatitis B virus-induced tubular epithelial-myofibroblast transdifferentiation Redox regulation of the actin cytoskeleton and its role in the vascular system.Change in Nox4 expression is accompanied by changes in myogenic marker expression in differentiating C2C12 myoblasts.NOX4-derived reactive oxygen species limit fibrosis and inhibit proliferation of vascular smooth muscle cells in diabetic atherosclerosis.Smad-independent pathway involved in transforming growth factor β1-induced Nox4 expression and proliferation of endothelial cells.NADPH oxidases in Parkinson's disease: a systematic review.

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NADPH oxidase 4 mediates TGF-β-induced smooth muscle α-actin via p38MAPK and serum response factor

http://www.wikidata.org/entity/Q42588680

description

2010 nî lūn-bûn

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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2010年论文

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2010年论文

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name

NADPH oxidase 4 mediates TGF-β ...... MAPK and serum response factor

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NADPH oxidase 4 mediates TGF-β ...... MAPK and serum response factor

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type

label

NADPH oxidase 4 mediates TGF-β ...... MAPK and serum response factor

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NADPH oxidase 4 mediates TGF-β ...... MAPK and serum response factor

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prefLabel

NADPH oxidase 4 mediates TGF-β ...... MAPK and serum response factor

@en

NADPH oxidase 4 mediates TGF-β ...... MAPK and serum response factor

@nl

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J.FREERADBIOMED.2010.11.007

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Free Radical Biology and Medicine

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NADPH oxidase 4 mediates TGF-β ...... MAPK and serum response factor

@en

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Abel Martin-Garrido

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Alejandra San Martín

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Alicia N Lyle

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Anna Dikalova

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Bernard Lassègue

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Bonnie Seidel-Rogol

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P2860

Megakaryoblastic leukemia-1/2, a transcriptional co-activator of serum response factor, is required for skeletal myogenic differentiation

Constitutive induction of p-Erk1/2 accompanied by reduced activities of protein phosphatases 1 and 2A and MKP3 due to reactive oxygen species during cellular senescence

Distinct roles of Nox1 and Nox4 in basal and angiotensin II-stimulated superoxide and hydrogen peroxide production

Megakaryoblastic leukemia factor-1 transduces cytoskeletal signals and induces smooth muscle cell differentiation from undifferentiated embryonic stem cells

TAB1: an activator of the TAK1 MAPKKK in TGF-beta signal transduction

Characterization of SAP-1, a protein recruited by serum response factor to the c-fos serum response element

Genetic characterization and cloning of mothers against dpp, a gene required for decapentaplegic function in Drosophila melanogaster.

A growth factor-induced kinase phosphorylates the serum response factor at a site that regulates its DNA-binding activity

Identification of a member of the MAPKKK family as a potential mediator of TGF-beta signal transduction

Identification of multiple SRF N-terminal phosphorylation sites affecting DNA binding properties

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354-362

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scholarly article

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10.1016/J.FREERADBIOMED.2010.11.007

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2

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50

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Kathy K. Griendling

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2010-11-11T00:00:00Z

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47790038

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21074607

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3032946

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