MRS of brain metabolite levels demonstrates the ability of scavenging of excess brain glutamate to protect against nerve agent induced seizures

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Glutamate, T cells and multiple sclerosis.The Glutamate-Glutamine Cycle in Epilepsy.Inhibitor of Endocannabinoid Deactivation Protects Against In Vitro and In Vivo Neurotoxic Effects of Paraoxon.

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MRS of brain metabolite levels demonstrates the ability of scavenging of excess brain glutamate to protect against nerve agent induced seizures

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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MRS of brain metabolite levels ...... t nerve agent induced seizures

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MRS of brain metabolite levels ...... t nerve agent induced seizures

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MRS of brain metabolite levels ...... t nerve agent induced seizures

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MRS of brain metabolite levels ...... t nerve agent induced seizures

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MRS of brain metabolite levels ...... t nerve agent induced seizures

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MRS of brain metabolite levels ...... t nerve agent induced seizures

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P1476

MRS of brain metabolite levels ...... t nerve agent induced seizures

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Arik Markovich

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David Mirelman

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Inbal E Biton

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P2860

1H-MRS can monitor metabolites changes of lateral intraventricular BDNF infusion into a mouse model of Alzheimer's disease in vivo.

Early experience of translating pH-weighted MRI to image human subjects at 3 Tesla.

Review of the value of gacyclidine (GK-11) as adjuvant medication to conventional treatments of organophosphate poisoning: primate experiments mimicking various scenarios of military or terrorist attack by soman.

Development of a prolonged calcium plateau in hippocampal neurons in rats surviving status epilepticus induced by the organophosphate diisopropylfluorophosphate.

Oxygen-inducible glutamate oxaloacetate transaminase as protective switch transforming neurotoxic glutamate to metabolic fuel during acute ischemic stroke.

Neuroprotection by glutamate oxaloacetate transaminase in ischemic stroke: an experimental study

Homeostasis of glutamate in brain fluids: an accelerated brain-to-blood efflux of excess glutamate is produced by blood glutamate scavenging and offers protection from neuropathologies.

Seizure activity post organophosphate exposure.

Glutamate efflux at the blood-brain barrier: cellular mechanisms and potential clinical relevance.

Association between pH-weighted endogenous amide proton chemical exchange saturation transfer MRI and tissue lactic acidosis during acute ischemic stroke.

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3226-3236

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scholarly article

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10.3390/IJMS16023226

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English

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Angela Ruban

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2015-02-02T00:00:00Z

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272081772

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25648322

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4346891

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MRS of brain metabolite levels demonstrates the ability of scavenging of excess brain glutamate to protect against nerve agent induced seizures

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P2860

MRS of brain metabolite levels demonstrates the ability of scavenging of excess brain glutamate to protect against nerve agent induced seizures

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P1476

P2093

P2860

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