about
Gender differences in non-ischemic myocardial remodeling: are they due to estrogen modulation of cardiac mast cells and/or membrane type 1 matrix metalloproteinaseSympathetic nervous system modulation of inflammation and remodeling in the hypertensive heartInhibition of matrix metalloproteinase activity prevents increases in myocardial tumor necrosis factor-alpha.Interleukin 6 mediates myocardial fibrosis, concentric hypertrophy, and diastolic dysfunction in rats.Cardiac mast cells: the centrepiece in adverse myocardial remodelling.Substance P induces adverse myocardial remodelling via a mechanism involving cardiac mast cells.Reversal of cardiac dysfunction by selective ET-A receptor antagonism.Protection from adverse myocardial remodeling secondary to chronic volume overload in mast cell deficient ratsEstrogenic modulation of inflammation-related genes in male rats following volume overloadPrevention of adverse cardiac remodeling to volume overload in female rats is the result of an estrogen-altered mast cell phenotype.Tryptase/Protease-activated receptor 2 interactions induce selective mitogen-activated protein kinase signaling and collagen synthesis by cardiac fibroblastsStem cell factor is responsible for the rapid response in mature mast cell density in the acutely stressed heart.Could interferon-gamma be a therapeutic target for treating heart failure?TNF-alpha inhibition attenuates adverse myocardial remodeling in a rat model of volume overload.The role of neuropeptides in adverse myocardial remodeling and heart failure.Novel Omega-3 Fatty Acid Epoxygenase Metabolite Reduces Kidney Fibrosis.Regulation of matrix metalloproteinases is at the heart of myocardial remodeling.Cardiac mast cells mediate left ventricular fibrosis in the hypertensive rat heart.The convergence of ancient chinese medicine with modern therapeutics to prevent cardiac fibrosis.Mast Cells: Key Contributors to Cardiac Fibrosis.Targeting substance P and relaxin: A future combination therapy approach for heart failure?Cardiovascular changes during maturation and ageing in male and female spontaneously hypertensive rats.Response of cardiac mast cells to atrial natriuretic peptide.Inhibition of matrix metalloproteinase activity by ACE inhibitors prevents left ventricular remodeling in a rat model of heart failure.Rosuvastatin attenuates hypertension-induced cardiovascular remodeling without affecting blood pressure in DOCA-salt hypertensive ratsAlterations in cardiac structure and function in a murine model of chronic alcohol consumptionSubstance P and the neurokinin-1 receptor in the ischaemic heart: Two sides to the coinThe Diabetic Cardiac Fibroblast: Mechanisms Underlying Phenotype and FunctionSubstance P-mediated cardiac mast cell activation: An in vitro studyDoxorubicin-Induced Myocardial Fibrosis Involves the Neurokinin-1 Receptor and Direct Effects on Cardiac FibroblastsUnderstanding the Complex Roles of Substance P in the Diseased HeartRegulation of Cardiac Mast Cell Maturation and Function by the Neurokinin-1 Receptor in the Fibrotic Heart
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P50
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hulumtues
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հետազոտող
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Scott Levick
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Scott Levick
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Scott Levick
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Scott Levick
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Scott Levick
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Scott Levick
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Scott Levick
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Scott Levick
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Scott Levick
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Scott Levick
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Scott Levick
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Scott Levick
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Scott Levick
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Scott Levick
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Scott Levick
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