Epigenetic switches in clag3 genes mediate blasticidin S resistance in malaria parasites.
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A genomic and evolutionary approach reveals non-genetic drug resistance in malariaSynergistic Malaria Parasite Killing by Two Types of Plasmodial Surface Anion Channel InhibitorsMalaria Parasite CLAG3, a Protein Linked to Nutrient Channels, Participates in High Molecular Weight Membrane-Associated Complexes in the Infected ErythrocyteProteolysis at a specific extracellular residue implicates integral membrane CLAG3 in malaria parasite nutrient channelsA kinetic fluorescence assay reveals unusual features of Ca⁺⁺ uptake in Plasmodium falciparum-infected erythrocytes.High guanidinium permeability reveals dehydration-dependent ion selectivity in the plasmodial surface anion channel.The conserved clag multigene family of malaria parasites: essential roles in host-pathogen interactionHow do antimalarial drugs reach their intracellular targets?A CLAG3 mutation in an amphipathic transmembrane domain alters malaria parasite nutrient channels and confers leupeptin resistance.Epigenetic Roulette in Blood Stream Plasmodium: Gambling on Sex.Mutations in the Plasmodium falciparum chloroquine resistance transporter, PfCRT, enlarge the parasite's food vacuole and alter drug sensitivities.New Assays to Characterise Growth-Related Phenotypes of Plasmodium falciparum Reveal Variation in Density-Dependent Growth Inhibition between Parasite Lines.Role of Plasmodium vivax Duffy-binding protein 1 in invasion of Duffy-null Africans.Indels, structural variation, and recombination drive genomic diversity in Plasmodium falciparum.An essential dual-function complex mediates erythrocyte invasion and channel-mediated nutrient uptake in malaria parasites.Why do malaria parasites increase host erythrocyte permeability?Deciphering the principles that govern mutually exclusive expression of Plasmodium falciparum clag3 genes.Malaria Epigenetics.Expression of the Plasmodium falciparum Clonally Variant clag3 Genes in Human Infections.Antiplasmodial activities of dyes against Plasmodium falciparum asexual and sexual stages: Contrasted uptakes of triarylmethanes Brilliant green, Green S (E142), and Patent Blue V (E131) by erythrocytes.Generating conditional gene knockouts in Plasmodium - a toolkit to produce stable DiCre recombinase-expressing parasite lines using CRISPR/Cas9.An upstream open reading frame (uORF) signals for cellular localization of the virulence factor implicated in pregnancy associated malaria.CLAG3 Self-Associates in Malaria Parasites and Quantitatively Determines Nutrient Uptake Channels at the Host Membrane.Malaria parasite proteins involved in nutrient channels at the host erythrocyte membrane: advances and questions for future researchEpigenetics of Malaria Parasites
P2860
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P2860
Epigenetic switches in clag3 genes mediate blasticidin S resistance in malaria parasites.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Epigenetic switches in clag3 genes mediate blasticidin S resistance in malaria parasites.
@en
Epigenetic switches in clag3 genes mediate blasticidin S resistance in malaria parasites.
@nl
type
label
Epigenetic switches in clag3 genes mediate blasticidin S resistance in malaria parasites.
@en
Epigenetic switches in clag3 genes mediate blasticidin S resistance in malaria parasites.
@nl
prefLabel
Epigenetic switches in clag3 genes mediate blasticidin S resistance in malaria parasites.
@en
Epigenetic switches in clag3 genes mediate blasticidin S resistance in malaria parasites.
@nl
P2093
P2860
P356
P1476
Epigenetic switches in clag3 genes mediate blasticidin S resistance in malaria parasites.
@en
P2093
Lindsey M Altenhofen
Manuel Llinás
Núria Rovira-Graells
Sofía Mira-Martínez
P2860
P304
P356
10.1111/CMI.12162
P577
2013-07-19T00:00:00Z