A dynamic notch injury response activates epicardium and contributes to fibrosis repair. - Wikidata - awgldk - TriplyDB

A dynamic notch injury response activates epicardium and contributes to fibrosis repair.

A dynamic notch injury response activates epicardium and contributes to fibrosis repair.

http://www.wikidata.org/entity/Q42620629

about

Epicardial Epithelial-to-Mesenchymal Transition in Heart Development and Disease Coordinating tissue interactions: Notch signaling in cardiac development and disease Endocardial and epicardial epithelial to mesenchymal transitions in heart development and disease Impact of notch signaling on inflammatory responses in cardiovascular disorders Role of Notch signaling in the mammalian heart MicroRNAs in heart failure: Small molecules with major impact Cardiac fibrosis in myocardial infarction-from repair and remodeling to regeneration The role of notch in the cardiovascular system: potential adverse effects of investigational notch inhibitors miR-21 promotes fibrogenic epithelial-to-mesenchymal transition of epicardial mesothelial cells involving Programmed Cell Death 4 and Sprouty-1 Epicardial YAP/TAZ orchestrate an immunosuppressive response following myocardial infarction Cardiac fibroblast in development and wound healing.CDC42 is required for epicardial and pro-epicardial development by mediating FGF receptor trafficking to the plasma membrane Heterocellular communication in the heart: electron tomography of telocyte-myocyte junctions.Telocytes and putative stem cells in the lungs: electron microscopy, electron tomography and laser scanning microscopy.The Notch pathway controls fibrotic and regenerative repair in the adult heart.Cardiac telocytes - their junctions and functional implications.The bHLH transcription factor Tcf21 is required for lineage-specific EMT of cardiac fibroblast progenitors.Notch signaling in descending thoracic aortic aneurysm and dissection Cardiotoxicity due to cancer therapy.Identification of telocytes in skeletal muscle interstitium: implication for muscle regeneration The cytoplasmic domain of TGFβR3 through its interaction with the scaffolding protein, GIPC, directs epicardial cell behavior Epithelial-to-mesenchymal and endothelial-to-mesenchymal transition: from cardiovascular development to disease.Notch signaling and cardiac repair.Jagged1 intracellular domain-mediated inhibition of Notch1 signalling regulates cardiac homeostasis in the postnatal heart.The Living Scar--Cardiac Fibroblasts and the Injured Heart.Cardiac Fibrosis: The Fibroblast Awakens Experimental acute myocardial infarction: telocytes involvement in neo-angiogenesis.Differential expression of embryonic epicardial progenitor markers and localization of cardiac fibrosis in adult ischemic injury and hypertensive heart disease.Notch signaling regulates cardiomyocyte proliferation during zebrafish heart regeneration.The epicardium as a candidate for heart regeneration.Cell-cell interaction in the heart via Wnt/β-catenin pathway after cardiac injury.Notch signaling mediated by TGF-β/Smad pathway in concanavalin A-induced liver fibrosis in rats.Epicardial progenitor cells in cardiac development and regeneration.New insights into the mechanism of notch signalling in fibrosis.Cardiac fibroblasts, fibrosis and extracellular matrix remodeling in heart disease.Recent advances in heart regeneration.The cardiac hypoxic niche: emerging role of hypoxic microenvironment in cardiac progenitors.Targeting cardiac fibroblasts to treat fibrosis of the heart: focus on HDACs.Making it stick: chasing the optimal stem cells for cardiac regeneration.Generation of cardiac progenitor cells through epicardial to mesenchymal transition.

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P2860

A dynamic notch injury response activates epicardium and contributes to fibrosis repair.

http://www.wikidata.org/entity/Q42620629

description

2010 nî lūn-bûn

@nan

2010年の論文

@ja

2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

2010年论文

@zh

2010年论文

@zh-cn

name

A dynamic notch injury response activates epicardium and contributes to fibrosis repair.

@en

A dynamic notch injury response activates epicardium and contributes to fibrosis repair.

@nl

type

label

A dynamic notch injury response activates epicardium and contributes to fibrosis repair.

@en

A dynamic notch injury response activates epicardium and contributes to fibrosis repair.

@nl

prefLabel

A dynamic notch injury response activates epicardium and contributes to fibrosis repair.

@en

A dynamic notch injury response activates epicardium and contributes to fibrosis repair.

@nl

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P356

CIRCRESAHA.110.233262

P1433

Circulation Research

P1476

A dynamic notch injury response activates epicardium and contributes to fibrosis repair.

@en

P2093

Eric N Olson

http://www.w3.org/2001/XMLSchema#string

Jamie L Russell

http://www.w3.org/2001/XMLSchema#string

Jay W Schneider

http://www.w3.org/2001/XMLSchema#string

Joseph A Hill

http://www.w3.org/2001/XMLSchema#string

Nicholas R Gaiano

http://www.w3.org/2001/XMLSchema#string

Sean C Goetsch

http://www.w3.org/2001/XMLSchema#string

P2860

Epithelial-mesenchymal transition and its implications for fibrosis

Concise review: mesenchymal stem/multipotent stromal cells: the state of transdifferentiation and modes of tissue repair--current views

Activation of Notch-mediated protective signaling in the myocardium

Capsulin: a novel bHLH transcription factor expressed in epicardial progenitors and mesenchyme of visceral organs

Complex networks orchestrate epithelial-mesenchymal transitions

Dll4 signalling through Notch1 regulates formation of tip cells during angiogenesis

Endothelial-to-mesenchymal transition contributes to cardiac fibrosis

Enhanced differentiation of human embryonic stem cells into cardiomyocytes by combining hanging drop culture and 5-azacytidine treatment.

Activation of Notch signaling pathway precedes heart regeneration in zebrafish.

Evidence from a genetic fate-mapping study that stem cells refresh adult mammalian cardiomyocytes after injury.

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51-59

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scholarly article

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10.1161/CIRCRESAHA.110.233262

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1

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108

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2010-11-24T00:00:00Z

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21106942

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3042596

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