Defective smooth muscle regulation in cGMP kinase I-deficient mice. - Wikidata - awgldk - TriplyDB

Defective smooth muscle regulation in cGMP kinase I-deficient mice.

Defective smooth muscle regulation in cGMP kinase I-deficient mice.

http://www.wikidata.org/entity/Q42645524

about

Presynaptically localized cyclic GMP-dependent protein kinase 1 is a key determinant of spinal synaptic potentiation and pain hypersensitivity Regulation of human endothelial cell focal adhesion sites and migration by cGMP-dependent protein kinase I Deletion of the protein kinase A/protein kinase G target SMTNL1 promotes an exercise-adapted phenotype in vascular smooth muscle Impairment of LTD and cerebellar learning by Purkinje cell-specific ablation of cGMP-dependent protein kinase I Mechanisms of Vascular Smooth Muscle Contraction and the Basis for Pharmacologic Treatment of Smooth Muscle Disorders Genetic modification of hypertension by sGCα1 Extending the translational potential of targeting NO/cGMP-regulated pathways in the CVS The C. elegans cGMP-dependent protein kinase EGL-4 regulates nociceptive behavioral sensitivity cGMP-dependent protein kinase type I is implicated in the regulation of the timing and quality of sleep and wakefulness Structural Basis for Cyclic-Nucleotide Selectivity and cGMP-Selective Activation of PKG I Crystal Structure of the cGMP-dependent Protein Kinase II Leucine Zipper and Rab11b Protein Complex Reveals Molecular Details of G-kinase-specific Interactions Serine 19 of human 6-pyruvoyltetrahydropterin synthase is phosphorylated by cGMP protein kinase II cGMP-dependent protein kinase I beta physically and functionally interacts with the transcriptional regulator TFII-I Phosphorylation and regulation of G-protein-activated phospholipase C-beta 3 by cGMP-dependent protein kinases Formin homology domain protein (FHOD1) is a cyclic GMP-dependent protein kinase I-binding protein and substrate in vascular smooth muscle cells Functional coupling between nitric oxide synthesis and VIP release within enteric nerve terminals of the rat: involvement of protein kinase G and phosphodiesterase 5 Guanosine 3': 5'-cyclic monophosphate-dependent pathway alterations in ventricular cardiomyocytes of spontaneously hypertensive rats Nitric oxide activation of p38 mitogen-activated protein kinase in 293T fibroblasts requires cGMP-dependent protein kinase Imaging of nitric oxide in nitrergic neuromuscular neurotransmission in the gut Spare guanylyl cyclase NO receptors ensure high NO sensitivity in the vascular system IRAG is essential for relaxation of receptor-triggered smooth muscle contraction by cGMP kinase Atrial natriuretic peptide induces natriuretic peptide receptor-cGMP-dependent protein kinase interaction Binding and phosphorylation of a novel male germ cell-specific cGMP-dependent protein kinase-anchoring protein by cGMP-dependent protein kinase Ialpha RhoA-mediated Ca2+ sensitization in erectile function.Stress-dependent dilated cardiomyopathy in mice with cardiomyocyte-restricted inactivation of cyclic GMP-dependent protein kinase I Smooth muscle specific Rac1 deficiency induces hypertension by preventing p116RIP3-dependent RhoA inhibition.Highly specific, membrane-permeant peptide blockers of cGMP-dependent protein kinase Ialpha inhibit NO-induced cerebral dilation.Airway smooth muscle relaxation results from a reduction in the frequency of Ca2+ oscillations induced by a cAMP-mediated inhibition of the IP3 receptor.cGMP-dependent protein kinase I is crucial for angiogenesis and postnatal vasculogenesis The Rho/Rac exchange factor Vav2 controls nitric oxide-dependent responses in mouse vascular smooth muscle cells.Adiponectin resistance and vascular dysfunction in the hyperlipidemic state Responses to enteric motor neurons in the gastric fundus of mice with reduced intramuscular interstitial cells of cajal Inducible knockout mutagenesis reveals compensatory mechanisms elicited by constitutive BK channel deficiency in overactive murine bladder.Lessons from genetically engineered animal models. IV. Nitric oxide synthase gene knockout mice.Cardiac hypertrophy is not amplified by deletion of cGMP-dependent protein kinase I in cardiomyocytes.Headache-type adverse effects of NO donors: vasodilation and beyond Hypoxia induces severe right ventricular dilatation and infarction in heme oxygenase-1 null mice.The vasodilator-stimulated phosphoprotein (VASP) is involved in cGMP- and cAMP-mediated inhibition of agonist-induced platelet aggregation, but is dispensable for smooth muscle function.Genes involved in vasoconstriction and vasodilation system affect salt-sensitive hypertension.Toxoplasma gondii cyclic GMP-dependent kinase: chemotherapeutic targeting of an essential parasite protein kinase.

P2860

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P2860

Defective smooth muscle regulation in cGMP kinase I-deficient mice.

http://www.wikidata.org/entity/Q42645524

description

1998 nî lūn-bûn

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1998年の論文

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1998年学术文章

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1998年学术文章

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1998年学术文章

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1998年学术文章

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1998年学术文章

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1998年學術文章

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1998年學術文章

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1998年學術文章

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name

Defective smooth muscle regulation in cGMP kinase I-deficient mice.

@en

Defective smooth muscle regulation in cGMP kinase I-deficient mice.

@nl

type

label

Defective smooth muscle regulation in cGMP kinase I-deficient mice.

@en

Defective smooth muscle regulation in cGMP kinase I-deficient mice.

@nl

prefLabel

Defective smooth muscle regulation in cGMP kinase I-deficient mice.

@en

Defective smooth muscle regulation in cGMP kinase I-deficient mice.

@nl

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The EMBO Journal

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Defective smooth muscle regulation in cGMP kinase I-deficient mice.

@en

P2093

Andersson KE

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Aszódi A

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Fässler R

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Hirneiss C

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Hofmann F

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Klatt P

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Korth M

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Krombach F

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Massberg S

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Mayerhofer A

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P2860

Molecular cloning and predicted full-length amino acid sequence of the type I beta isozyme of cGMP-dependent protein kinase from human placenta. Tissue distribution and developmental changes in rat

The protein kinase family: conserved features and deduced phylogeny of the catalytic domains

Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factor

Consequences of lack of beta 1 integrin gene expression in mice

The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholine

Endothelium-derived relaxing factor produced and released from artery and vein is nitric oxide

Nitroxergic nerves mediate vagally induced relaxation in the isolated stomach of the guinea pig.

Signal transduction and regulation in smooth muscle.

Adenosine activates ATP-sensitive potassium channels in arterial myocytes via A2 receptors and cAMP-dependent protein kinase

cGMP-dependent protein kinase mediates the reduction of Ca2+ by cAMP in vascular smooth muscle cells.

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3045-3051

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11

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9606187

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