about
Cerebral oedema is not responsible for motor or cognitive deficits in rats with hepatic encephalopathy.Proteome analysis of primary neurons and astrocytes from rat cerebellum.Infliximab reduces Zaprinast-induced retinal degeneration in cultures of porcine retina.Novel mutations in the USH1C gene in Usher syndrome patients.Targeted next generation sequencing for molecular diagnosis of Usher syndrome.Study of USH1 splicing variants through minigenes and transcript analysis from nasal epithelial cells.Altered antioxidant-oxidant status in the aqueous humor and peripheral blood of patients with retinitis pigmentosa.Altered modulation of soluble guanylate cyclase by nitric oxide in patients with liver disease.Mutational screening of the USH2A gene in Spanish USH patients reveals 23 novel pathogenic mutationscGMP-Phosphodiesterase Inhibition Prevents Hypoxia-Induced Cell Death Activation in Porcine Retinal ExplantsAltered modulation of motor activity by group I metabotropic glutamate receptors in the nucleus accumbens in hyperammonemic rats.Brain regional alterations in the modulation of the glutamate-nitric oxide-cGMP pathway in liver cirrhosis. Role of hyperammonemia and cell types involved.Two novel disease-causing mutations in the CLRN1 gene in patients with Usher syndrome type 3.Control of brain glutamine synthesis by NMDA receptors.NMDA receptors in hyperammonemia and hepatic encephalopathy.Brain cholinergic impairment in liver failure.Pharmacological manipulation of cyclic GMP levels in brain restores learning ability in animal models of hepatic encephalopathy: therapeutic implications.Role of NMDA receptors in acute liver failure and ammonia toxicity: therapeutical implications.Cyclic GMP pathways in hepatic encephalopathy. Neurological and therapeutic implications.Adalimumab Reduces Photoreceptor Cell Death in A Mouse Model of Retinal Degeneration.Phosphate-activated glutaminase activity is enhanced in brain, intestine and kidneys of rats following portacaval anastomosisPolychlorinated biphenyls PCB 153 and PCB 126 impair the glutamate-nitric oxide-cGMP pathway in cerebellar neurons in culture by different mechanisms.Altered modulation of soluble guanylate cyclase in lymphocytes from patients with liver disease.Hyperammonemia induces neuroinflammation that contributes to cognitive impairment in rats with hepatic encephalopathy.Chronic hyperammonemia induces tonic activation of NMDA receptors in cerebellum.cGMP modulates stem cells differentiation to neurons in brain in vivo.Selective regional alterations in the content or distribution of neuronal and glial cytoskeletal proteins in brain of rats chronically exposed to 2,5-hexanedione.Oral administration of sildenafil restores learning ability in rats with hyperammonemia and with portacaval shunts.Bile duct ligation plus hyperammonemia in rats reproduces the alterations in the modulation of soluble guanylate cyclase by nitric oxide in brain of cirrhotic patients.cGMP modulates stem cells differentiation to neurons in brain in vivo pathological implications.Mechanisms of cognitive alterations in hyperammonemia and hepatic encephalopathy: therapeutical implications.Neuroinflammation contributes to hypokinesia in rats with hepatic encephalopathy: ibuprofen restores its motor activity.Haemodynamic effects of long-term administration of sildenafil in normotensive pregnant and non-pregnant rats.Neurons exposed to ammonia reproduce the differential alteration in nitric oxide modulation of guanylate cyclase in the cerebellum and cortex of patients with liver cirrhosis.Acute liver failure-induced death of rats is delayed or prevented by blocking NMDA receptors in brain.Chronic hyperammonemia reduces the activity of neuronal nitric oxide synthase in cerebellum by altering its localization and increasing its phosphorylation by calcium-calmodulin kinase II.HIF-1α stabilization reduces retinal degeneration in a mouse model of retinitis pigmentosa.Inflammation and hepatic encephalopathy: ibuprofen restores learning ability in rats with portacaval shunts.Hyperammonaemia alters the mechanisms by which metabotropic glutamate receptors in nucleus accumbens modulate motor function.Glutamate-induced activation of nitric oxide synthase is impaired in cerebral cortex in vivo in rats with chronic liver failure.
P50
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P50
description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Regina Rodrigo
@ast
Regina Rodrigo
@en
Regina Rodrigo
@es
Regina Rodrigo
@nl
Regina Rodrigo
@sl
type
label
Regina Rodrigo
@ast
Regina Rodrigo
@en
Regina Rodrigo
@es
Regina Rodrigo
@nl
Regina Rodrigo
@sl
prefLabel
Regina Rodrigo
@ast
Regina Rodrigo
@en
Regina Rodrigo
@es
Regina Rodrigo
@nl
Regina Rodrigo
@sl
P1053
I-1868-2015
P106
P21
P31
P3829
P496
0000-0001-5875-986X