Glucocorticoid receptor dimerization is required for proper recovery of LPS-induced inflammation, sickness behavior and metabolism in mice. - Wikidata - awgldk - TriplyDB

Glucocorticoid receptor dimerization is required for proper recovery of LPS-induced inflammation, sickness behavior and metabolism in mice.

Glucocorticoid receptor dimerization is required for proper recovery of LPS-induced inflammation, sickness behavior and metabolism in mice.

http://www.wikidata.org/entity/Q42673840

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PKR deficiency alters E. coli-induced sickness behaviors but does not exacerbate neuroimmune responses or bacterial load A Quantitative Study of Internal and External Interactions of Homodimeric Glucocorticoid Receptor Using Fluorescence Cross-Correlation Spectroscopy in a Live Cell.Toll-like receptor 4: innate immune regulator of neuroimmune and neuroendocrine interactions in stress and major depressive disorder Dissociation between sickness behavior and emotionality during lipopolysaccharide challenge in lymphocyte deficient Rag2(-/-) mice.Genomic redistribution of GR monomers and dimers mediates transcriptional response to exogenous glucocorticoid in vivo.Corticosteroids Mediate Heart Failure-Induced Depression through Reduced σ1-Receptor Expression.Endogenous hepatic glucocorticoid receptor signaling coordinates sex-biased inflammatory gene expression.The HPA - Immune Axis and the Immunomodulatory Actions of Glucocorticoids in the Brain.Inflammatory biomarker profiles of mental disorders and their relation to clinical, social and lifestyle factors.Molecular Actions of Glucocorticoids in Cartilage and Bone During Health, Disease, and Steroid Therapy.Monitoring Pathogen-Induced Sickness in Mice and Rats.Chronic Physical Stress Does Not Interact with Epstein-Barr Virus (EBV)-Encoded Dutpase to Alter the Sickness Response Anti-inflammatory Effect of Astaxanthin on the Sickness Behavior Induced by Diabetes Mellitus.Repeated lipopolysaccharide exposure modifies immune and sickness behaviour response in an animal model of chronic inflammation.Corticosterone Preexposure Increases NF-κB Translocation and Sensitizes IL-1β Responses in BV2 Microglia-Like Cells.Glucocorticoid Receptor Binding Induces Rapid and Prolonged Large-Scale Chromatin Decompaction at Multiple Target Loci.A screening assay for Selective Dimerizing Glucocorticoid Receptor Agonists and Modulators (SEDIGRAM) that are effective against acute inflammation Time and frequency dependent changes in resting state EEG functional connectivity following lipopolysaccharide challenge in rats

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P2860

Glucocorticoid receptor dimerization is required for proper recovery of LPS-induced inflammation, sickness behavior and metabolism in mice.

http://www.wikidata.org/entity/Q42673840

description

2012 nî lūn-bûn

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Glucocorticoid receptor dimeri ...... havior and metabolism in mice.

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Glucocorticoid receptor dimeri ...... havior and metabolism in mice.

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Glucocorticoid receptor dimeri ...... havior and metabolism in mice.

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Glucocorticoid receptor dimeri ...... havior and metabolism in mice.

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Glucocorticoid receptor dimeri ...... havior and metabolism in mice.

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Glucocorticoid receptor dimeri ...... havior and metabolism in mice.

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Molecular Psychiatry

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Glucocorticoid receptor dimeri ...... havior and metabolism in mice.

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B D Revenis

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B E Ballard

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E Belyavskaya

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E M Sternberg

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L H Tonelli

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M N Silverman

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P2860

Immune modulation of the hypothalamic-pituitary-adrenal (HPA) axis during viral infection

Cytokines sing the blues: inflammation and the pathogenesis of depression

From inflammation to sickness and depression: when the immune system subjugates the brain

Separating transrepression and transactivation: a distressing divorce for the glucocorticoid receptor?

Tumor necrosis factor (TNF)-mediated neuroprotection against glutamate-induced excitotoxicity is enhanced by N-methyl-D-aspartate receptor activation. Essential role of a TNF receptor 2-mediated phosphatidylinositol 3-kinase-dependent NF-kappa B pat

How do glucocorticoids influence stress responses? Integrating permissive, suppressive, stimulatory, and preparative actions

Inflammation and its discontents: the role of cytokines in the pathophysiology of major depression

Enhancing versus suppressive effects of stress on immune function: implications for immunoprotection and immunopathology

Should Depressive Syndromes Be Reclassified as "Metabolic Syndrome Type II"?

Fever and hypothermia in systemic inflammation: recent discoveries and revisions.

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1006-1017

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10.1038/MP.2012.131

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Partha Mukhopadhyay

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