Kinetics and peptide dependency of the binding of the inhibitory NK receptor CD94/NKG2-A and the activating receptor CD94/NKG2-C to HLA-E.
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HLA-E allelic variants. Correlating differential expression, peptide affinities, crystal structures, and thermal stabilitiesHuman CD8 T lymphocytes recognize Mycobacterium tuberculosis antigens presented by HLA-E during active tuberculosis and express type 2 cytokinesStructural basis for NKG2A/CD94 recognition of HLA-E.Peptide binding characteristics of the non-classical class Ib MHC molecule HLA-E assessed by a recombinant random peptide approachComparative genomics of natural killer cell receptor gene clusters.Current status and perspectives in translational biomarker research for PD-1/PD-L1 immune checkpoint blockade therapyEffects of Peptide on NK cell-mediated MHC I recognitionHLA-dependent tumour development: a role for tumour associate macrophages?Crystal structure of a lectin-like natural killer cell receptor bound to its MHC class I ligandCD94-NKG2A recognition of human leukocyte antigen (HLA)-E bound to an HLA class I leader sequenceRequirement of the proteasome for the trimming of signal peptide-derived epitopes presented by the nonclassical major histocompatibility complex class I molecule HLA-EDirect binding of beta-arrestins to two distinct intracellular domains of the delta opioid receptorThe nonpolymorphic MHC Qa-1b mediates CD8+ T cell surveillance of antigen-processing defectsTransgenic expression of HLA-E single chain trimer protects porcine endothelial cells against human natural killer cell-mediated cytotoxicityExclusion of lipid rafts and decreased mobility of CD94/NKG2A receptors at the inhibitory NK cell synapse.Analysis of Qa-1(b) peptide binding specificity and the capacity of CD94/NKG2A to discriminate between Qa-1-peptide complexesEpstein-Barr virus peptide presented by HLA-E is predominantly recognized by CD8(bright) cells in multiple sclerosis patientsDevelopment and function of CD94-deficient natural killer cellsInnate immune natural killer cells and their role in HIV and SIV infection.On guard--activating NK cell receptors.Mammalian non-classical major histocompatibility complex I and its receptors: Important contexts of gene, evolution, and immunity.Human natural killer cell receptors and signal transduction.IFN-gamma protects short-term ovarian carcinoma cell lines from CTL lysis via a CD94/NKG2A-dependent mechanismIFN-gamma suspends the killing license of anti-tumor CTLsImaging immune surveillance by T cells and NK cells.Activating signals dominate inhibitory signals in CD137L/IL-15 activated natural killer cells.Expansion of a unique CD57⁺NKG2Chi natural killer cell subset during acute human cytomegalovirus infectionHuman cytomegalovirus drives epigenetic imprinting of the IFNG locus in NKG2Chi natural killer cells.Activation of natural killer cells: underlying molecular mechanisms revealed.Cytomegalovirus reactivation after allogeneic transplantation promotes a lasting increase in educated NKG2C+ natural killer cells with potent function.A Conserved HIV-1-Derived Peptide Presented by HLA-E Renders Infected T-cells Highly Susceptible to Attack by NKG2A/CD94-Bearing Natural Killer Cells.Adaptive Natural Killer Cell and Killer Cell Immunoglobulin-Like Receptor-Expressing T Cell Responses are Induced by Cytomegalovirus and Are Associated with Protection against Cytomegalovirus Reactivation after Allogeneic Donor Hematopoietic Cell TrSmall intestinal CD8+TCRgammadelta+NKG2A+ intraepithelial lymphocytes have attributes of regulatory cells in patients with celiac disease.Major histocompatibility complex class I-recognizing receptors are disease risk genes in rheumatoid arthritis.A signal peptide derived from hsp60 binds HLA-E and interferes with CD94/NKG2A recognitionStress signals activate natural killer cells.Human cytomegalovirus (CMV)-induced memory-like NKG2C(+) NK cells are transplantable and expand in vivo in response to recipient CMV antigen.Recognition of the class Ib molecule Qa-1(b) by putative activating receptors CD94/NKG2C and CD94/NKG2E on mouse natural killer cellsThe immunoregulatory effects of Qa-1.HLA-B signal peptide polymorphism influences the rate of HIV-1 acquisition but not viral load
P2860
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P2860
Kinetics and peptide dependency of the binding of the inhibitory NK receptor CD94/NKG2-A and the activating receptor CD94/NKG2-C to HLA-E.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Kinetics and peptide dependenc ...... receptor CD94/NKG2-C to HLA-E.
@en
Kinetics and peptide dependenc ...... receptor CD94/NKG2-C to HLA-E.
@nl
type
label
Kinetics and peptide dependenc ...... receptor CD94/NKG2-C to HLA-E.
@en
Kinetics and peptide dependenc ...... receptor CD94/NKG2-C to HLA-E.
@nl
prefLabel
Kinetics and peptide dependenc ...... receptor CD94/NKG2-C to HLA-E.
@en
Kinetics and peptide dependenc ...... receptor CD94/NKG2-C to HLA-E.
@nl
P2093
P356
P1433
P1476
Kinetics and peptide dependenc ...... receptor CD94/NKG2-C to HLA-E.
@en
P2093
H T Reyburn
M López-Botet
M Valés-Gómez
R A Erskine
P304
P356
10.1093/EMBOJ/18.15.4250
P407
P577
1999-08-01T00:00:00Z