KCa3.1 Inhibition Switches the Astrocyte Phenotype during Astrogliosis Associated with Ischemic Stroke Via Endoplasmic Reticulum Stress and MAPK Signaling Pathways.

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KCa3.1 Inhibition Switches the Astrocyte Phenotype during Astrogliosis Associated with Ischemic Stroke Via Endoplasmic Reticulum Stress and MAPK Signaling Pathways.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

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2017年论文

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KCa3.1 Inhibition Switches the ...... s and MAPK Signaling Pathways.

@en

KCa3.1 Inhibition Switches the ...... s and MAPK Signaling Pathways.

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Item

label

KCa3.1 Inhibition Switches the ...... s and MAPK Signaling Pathways.

@en

KCa3.1 Inhibition Switches the ...... s and MAPK Signaling Pathways.

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prefLabel

KCa3.1 Inhibition Switches the ...... s and MAPK Signaling Pathways.

@en

KCa3.1 Inhibition Switches the ...... s and MAPK Signaling Pathways.

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P1476

KCa3.1 Inhibition Switches the ...... s and MAPK Signaling Pathways.

@en

P2093

Hongzhuan Chen

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Mengni Yi

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Tianjiao Wei

http://www.w3.org/2001/XMLSchema#string

Xiaoling Gao

http://www.w3.org/2001/XMLSchema#string

Zhihua Yu

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P2860

Up-regulation of the IKCa1 potassium channel during T-cell activation. Molecular mechanism and functional consequences

The Potassium Channel KCa3.1 Represents a Valid Pharmacological Target for Astrogliosis-Induced Neuronal Impairment in a Mouse Model of Alzheimer’s Disease

Targeted inhibition of KCa3.1 attenuates TGF-β-induced reactive astrogliosis through the Smad2/3 signaling pathway.

Reactive astrocytes in neural repair and protection.

Ion channels in the regulation of apoptosis.

The endoplasmic reticulum as an integrating signalling organelle: from neuronal signalling to neuronal death.

Molecular targets in cerebral ischemia for developing novel therapeutics.

The intermediate conductance calcium-activated potassium channel KCa3.1 regulates vascular smooth muscle cell proliferation via controlling calcium-dependent signaling

Inhibition of Intermediate-Conductance Calcium-Activated K Channel (KCa3.1) and Fibroblast Mitogenesis by α-Linolenic Acid and Alterations of Channel Expression in the Lysosomal Storage Disorders, Fabry Disease, and Niemann Pick C

Potassium channels Kv1.3 and KCa3.1 cooperatively and compensatorily regulate antigen-specific memory T cell functions.

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319

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scholarly article

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10.3389/FNCEL.2017.00319

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P478

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2017-10-12T00:00:00Z

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29075181

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P921

astrocyte

endoplasmic reticulum

P932

5643415

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