Herpes simplex virus-1 infection causes the secretion of a type I interferon-antagonizing protein and inhibits signaling at or before Jak-1 activation.
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Viral Inhibition of the IFN-Induced JAK/STAT Signalling Pathway: Development of Live Attenuated Vaccines by Mutation of Viral-Encoded IFN-AntagonistsType I interferon response and innate immune sensing of cancerIFI16 restricts HSV-1 replication by accumulating on the hsv-1 genome, repressing HSV-1 gene expression, and directly or indirectly modulating histone modificationsBioluminescent imaging reveals divergent viral pathogenesis in two strains of Stat1-deficient mice, and in αßγ interferon receptor-deficient mice.Inhibition of phosphorylated-STAT1 nuclear translocation and antiviral protein expression in human brain vascular adventitial fibroblasts infected with varicella-zoster virus.Reduced degranulation of NK cells in patients with frequently recurring herpes.HSV-2 inhibits type-I interferon signaling via multiple complementary and compensatory STAT2-associated mechanismsDuck enteritis virus UL54 is an IE protein primarily located in the nucleus.ICP27 protein encoded by bovine herpesvirus type 1 (bICP27) interferes with promoter activity of the bovine genes encoding beta interferon 1 (IFN-β1) and IFN-β3.Herpes simplex virus 1-encoded tegument protein VP16 abrogates the production of beta interferon (IFN) by inhibiting NF-κB activation and blocking IFN regulatory factor 3 to recruit its coactivator CBP.Herpes simplex virus 1 serine/threonine kinase US3 hyperphosphorylates IRF3 and inhibits beta interferon production.The many roles of the highly interactive HSV protein ICP27, a key regulator of infection.Type I interferons in regulation of mucosal immunity.Interferons as biomarkers and effectors: lessons learned from animal models.Herpesviruses: interfering innate immunity by targeting viral sensing and interferon pathways.Neurons versus herpes simplex virus: the innate immune interactions that contribute to a host-pathogen standoffInnate Immune Mechanisms and Herpes Simplex Virus Infection and Disease.Expression of IFN-Inducible Genes with Antiviral Function OAS1 and MX1 in Health and under Conditions of Recurrent Herpes Simplex Infection.A determinant of Sindbis virus neurovirulence enables efficient disruption of Jak/STAT signaling.Interplay between Janus Kinase/Signal Transducer and Activator of Transcription Signaling Activated by Type I Interferons and Viral Antagonism.Abortigenic but Not Neurotropic Equine Herpes Virus 1 Modulates the Interferon Antiviral Defense
P2860
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P2860
Herpes simplex virus-1 infection causes the secretion of a type I interferon-antagonizing protein and inhibits signaling at or before Jak-1 activation.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
Herpes simplex virus-1 infecti ...... at or before Jak-1 activation.
@en
Herpes simplex virus-1 infecti ...... at or before Jak-1 activation.
@nl
type
label
Herpes simplex virus-1 infecti ...... at or before Jak-1 activation.
@en
Herpes simplex virus-1 infecti ...... at or before Jak-1 activation.
@nl
prefLabel
Herpes simplex virus-1 infecti ...... at or before Jak-1 activation.
@en
Herpes simplex virus-1 infecti ...... at or before Jak-1 activation.
@nl
P2860
P1433
P1476
Herpes simplex virus-1 infecti ...... at or before Jak-1 activation.
@en
P2093
David M Knipe
Karen E Johnson
P2860
P356
10.1016/J.VIROL.2009.09.021
P407
P577
2009-10-31T00:00:00Z