Endoplasmic Reticulum Stress and Store-Operated Calcium Entry Contribute to Usnic Acid-Induced Toxicity in Hepatic Cells.
about
Endoplasmic reticulum stress and MAPK signaling pathway activation underlie leflunomide-induced toxicity in HepG2 Cells.Activation of the Nrf2 signaling pathway in usnic acid-induced toxicity in HepG2 cells.Endoplasmic Reticulum Stress Induction and ERK1/2 Activation Contribute to Nefazodone-Induced Toxicity in Hepatic Cells.Ca2+ Signaling and Cell Death Induced by Protriptyline in HepG2 Human Hepatoma Cells.Mitochondrial dysfunction induced by leflunomide and its active metabolite.
P2860
Endoplasmic Reticulum Stress and Store-Operated Calcium Entry Contribute to Usnic Acid-Induced Toxicity in Hepatic Cells.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
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2015年論文
@zh-tw
2015年论文
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2015年论文
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2015年论文
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name
Endoplasmic Reticulum Stress a ...... ced Toxicity in Hepatic Cells.
@en
Endoplasmic Reticulum Stress a ...... ced Toxicity in Hepatic Cells.
@nl
type
label
Endoplasmic Reticulum Stress a ...... ced Toxicity in Hepatic Cells.
@en
Endoplasmic Reticulum Stress a ...... ced Toxicity in Hepatic Cells.
@nl
prefLabel
Endoplasmic Reticulum Stress a ...... ced Toxicity in Hepatic Cells.
@en
Endoplasmic Reticulum Stress a ...... ced Toxicity in Hepatic Cells.
@nl
P2093
P2860
P356
P1476
Endoplasmic Reticulum Stress a ...... ced Toxicity in Hepatic Cells.
@en
P2093
William H Tolleson
Yuanfeng Wu
Zhuhong Zhang
P2860
P304
P356
10.1093/TOXSCI/KFV075
P50
P577
2015-04-13T00:00:00Z