Inhibiting TGF-β activity improves respiratory function in mdx mice. - Wikidata - awgldk - TriplyDB

Inhibiting TGF-β activity improves respiratory function in mdx mice.

Inhibiting TGF-β activity improves respiratory function in mdx mice.

http://www.wikidata.org/entity/Q42732727

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SIRT1: A Novel Target for the Treatment of Muscular Dystrophies Pre-clinical drug tests in the mdx mouse as a model of dystrophinopathies: an overview The role of fibrosis in Duchenne muscular dystrophy Overexpression of Latent TGFβ Binding Protein 4 in Muscle Ameliorates Muscular Dystrophy through Myostatin and TGFβ Modifier genes and their effect on Duchenne muscular dystrophy Phospholipase A2 in experimental allergic bronchitis: a lesson from mouse and rat models Biochemical and Functional Comparisons of mdx and Sgcg(-/-) Muscular Dystrophy Mouse Models.Targeting TGF-β Signaling by Antisense Oligonucleotide-mediated Knockdown of TGF-β Type I Receptor.Improving Reproducibility of Phenotypic Assessments in the DyW Mouse Model of Laminin-α2 Related Congenital Muscular Dystrophy.LTBP4 genotype predicts age of ambulatory loss in Duchenne muscular dystrophy.Pharmacologic management of Duchenne muscular dystrophy: target identification and preclinical trials Inflammatory monocytes promote progression of Duchenne muscular dystrophy and can be therapeutically targeted via CCR2.Early right ventricular fibrosis and reduction in biventricular cardiac reserve in the dystrophin-deficient mdx heart.A Wnt-TGFβ2 axis induces a fibrogenic program in muscle stem cells from dystrophic mice.Dimorphic effects of transforming growth factor-β signaling during aortic aneurysm progression in mice suggest a combinatorial therapy for Marfan syndrome Life or death by NFκB, Losartan promotes survival in dy2J/dy2J mouse of MDC1A.Chronic Dosing with Membrane Sealant Poloxamer 188 NF Improves Respiratory Dysfunction in Dystrophic Mdx and Mdx/Utrophin-/- Mice Genotype-Specific Interaction of Latent TGFβ Binding Protein 4 with TGFβ.Hypoxia-induced cardiac injury in dystrophic mice.Modifying muscular dystrophy through transforming growth factor-β.Conditional expression of TGF-β1 in skeletal muscles causes endomysial fibrosis and myofibers atrophy Coronary adventitial cells are linked to perivascular cardiac fibrosis via TGFβ1 signaling in the mdx mouse model of Duchenne muscular dystrophy.Prospect for pharmacological therapies to treat skeletal muscle dysfunction.Drug Discovery of Therapies for Duchenne Muscular Dystrophy.A healthy tension in translational research.Fibrillin-1 Regulates Skeletal Stem Cell Differentiation by Modulating TGFβ Activity Within the Marrow Niche.Muscle cell communication in development and repair.Andrographolide attenuates skeletal muscle dystrophy in mdx mice and increases efficiency of cell therapy by reducing fibrosis.Motor performance of young dystrophic mdx mice treated with long-circulating prednisolone liposomes.Excess SMAD signaling contributes to heart and muscle dysfunction in muscular dystrophy.Targeting latent TGFβ release in muscular dystrophy.ECM-Related Myopathies and Muscular Dystrophies: Pros and Cons of Protein Therapies.Biochemical and morphological alterations in the Achilles tendon of mdx mice.Therapeutic effects of mouse adipose-derived stem cells and losartan in the skeletal muscle of injured mdx mice.Non-Glycanated Biglycan and LTBP4: Leveraging the extracellular matrix for Duchenne Muscular Dystrophy therapeutics.The dual CCR2/CCR5 chemokine receptor antagonist Cenicriviroc reduces macrophage infiltration and disease severity in Duchenne muscular dystrophy (Dmdmdx-4Cv) mice.

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Inhibiting TGF-β activity improves respiratory function in mdx mice.

http://www.wikidata.org/entity/Q42732727

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2011 nî lūn-bûn

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name

Inhibiting TGF-β activity improves respiratory function in mdx mice.

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Inhibiting TGF-β activity improves respiratory function in mdx mice.

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type

label

Inhibiting TGF-β activity improves respiratory function in mdx mice.

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Inhibiting TGF-β activity improves respiratory function in mdx mice.

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prefLabel

Inhibiting TGF-β activity improves respiratory function in mdx mice.

@en

Inhibiting TGF-β activity improves respiratory function in mdx mice.

@nl

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J.AJPATH.2011.02.024

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The American Journal of Pathology

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Inhibiting TGF-β activity improves respiratory function in mdx mice.

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P2093

Alison McVie-Wylie

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Bruce M Wentworth

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Carol A Nelson

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Carol J Dinardo

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Jennifer A McCullough

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John P Leonard

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Kelly A Keefe

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Lindsay A Quigley

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Lorena Ceci

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Nicholas P Clayton

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P2860

Transforming growth factor beta 1 null mutation in mice causes excessive inflammatory response and early death

Primary structure of dystrophin-associated glycoproteins linking dystrophin to the extracellular matrix

TGF-beta inhibits muscle differentiation through functional repression of myogenic transcription factors by Smad3

Identification and characterization of a novel protein, periostin, with restricted expression to periosteum and periodontal ligament and increased expression by transforming growth factor beta

Dystrophin: the protein product of the Duchenne muscular dystrophy locus

Complete cloning of the duchenne muscular dystrophy (DMD) cDNA and preliminary genomic organization of the DMD gene in normal and affected individuals

Arsenic inhibits myogenic differentiation and muscle regeneration.

Losartan decreases cardiac muscle fibrosis and improves cardiac function in dystrophin-deficient mdx mice.

The dystrophin-glycoprotein complex, cellular signaling, and the regulation of cell survival in the muscular dystrophies.

Controversies about the function of dystrophin in muscle.

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51191682

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21641384

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