Rictor/mTORC2 loss in the Myf5 lineage reprograms brown fat metabolism and protects mice against obesity and metabolic disease
about
Impacts of Alternative Splicing Events on the Differentiation of AdipocytesThe Mechanistic Target of Rapamycin: The Grand ConducTOR of Metabolism and AgingThe Complex Roles of Mechanistic Target of Rapamycin in Adipocytes and Beyond.Acid sphingomyelinase deficiency in Western diet-fed mice protects against adipocyte hypertrophy and diet-induced liver steatosis.Emerging function of mTORC2 as a core regulator in glioblastoma: metabolic reprogramming and drug resistance.Highly selective in vivo labeling of subcutaneous white adipocyte precursors with Prx1-CremTOR is necessary for proper satellite cell activity and skeletal muscle regenerationTranscriptional and post-transcriptional control of adipocyte differentiation by Jumonji domain-containing protein 6.mTORC2 sustains thermogenesis via Akt-induced glucose uptake and glycolysis in brown adipose tissue.Recent Advances in Adipose mTOR Signaling and Function: Therapeutic ProspectsAdipose tissue mTORC2 regulates ChREBP-driven de novo lipogenesis and hepatic glucose metabolismEmerging Complexities in Adipocyte Origins and Identity.Heterogeneous nuclear ribonucleoprotein M associates with mTORC2 and regulates muscle differentiation.Akt kinase C-terminal modifications control activation loop dephosphorylation and enhance insulin responsemTORC1 and mTORC2 as regulators of cell metabolism in immunity.Adipocyte-specific deletion of mTOR inhibits adipose tissue development and causes insulin resistance in mice.Brown Fat AKT2 Is a Cold-Induced Kinase that Stimulates ChREBP-Mediated De Novo Lipogenesis to Optimize Fuel Storage and Thermogenesis.Selective and membrane-permeable small molecule inhibitors of nicotinamide N-methyltransferase reverse high fat diet-induced obesity in mice.Taming the Flames: Targeting White Adipose Tissue Browning in Hypermetabolic Conditions.Loss of Rictor in Monocyte/Macrophages Suppresses Their Proliferation and Viability Reducing Atherosclerosis in LDLR Null Mice.Adipocyte-specific deletion of Lkb1 and mTOR protects against high-fat diet induced obesity but results in insulin resistance.
P2860
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P2860
Rictor/mTORC2 loss in the Myf5 lineage reprograms brown fat metabolism and protects mice against obesity and metabolic disease
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
2014年论文
@zh
2014年论文
@zh-cn
name
Rictor/mTORC2 loss in the Myf5 ...... obesity and metabolic disease
@en
Rictor/mTORC2 loss in the Myf5 ...... obesity and metabolic disease
@nl
type
label
Rictor/mTORC2 loss in the Myf5 ...... obesity and metabolic disease
@en
Rictor/mTORC2 loss in the Myf5 ...... obesity and metabolic disease
@nl
prefLabel
Rictor/mTORC2 loss in the Myf5 ...... obesity and metabolic disease
@en
Rictor/mTORC2 loss in the Myf5 ...... obesity and metabolic disease
@nl
P2093
P2860
P50
P1433
P1476
Rictor/mTORC2 loss in the Myf5 ...... obesity and metabolic disease
@en
P2093
Amy J Wagers
Camila Martinez Calejman
Chien-Min Hung
David A Guertin
Simone Hettmer
P2860
P304
P356
10.1016/J.CELREP.2014.06.007
P577
2014-07-04T00:00:00Z