Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries. - Wikidata - awgldk - TriplyDB

Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.

Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.

http://www.wikidata.org/entity/Q42735742

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Endothelium-dependent smooth muscle hyperpolarization: do gap junctions provide a unifying hypothesis?Inducible endothelium-derived hyperpolarizing factor: role of the 15-lipoxygenase-EDHF pathway Further investigations into the endothelium-dependent hyperpolarizing effects of bradykinin and substance P in porcine coronary artery Blockade of chloride channels reveals relaxations of rat small mesenteric arteries to raised potassium Bradykinin attenuates the [Ca(2+)](i) response to angiotensin II of renal juxtamedullary efferent arterioles via an EDHF Suppression of K(+)-induced hyperpolarization by phenylephrine in rat mesenteric artery: relevance to studies of endothelium-derived hyperpolarizing factor Alterations in EDHF-mediated hyperpolarization and relaxation in mesenteric arteries of female rats in long-term deficiency of oestrogen and during oestrus cycle K+ currents underlying the action of endothelium-derived hyperpolarizing factor in guinea-pig, rat and human blood vessels Regulation of cellular communication by signaling microdomains in the blood vessel wall.Modulation of endothelial cell KCa3.1 channels during endothelium-derived hyperpolarizing factor signaling in mesenteric resistance arteries.Angiotensin II receptor blockade corrects altered expression of gap junctions in vascular endothelial cells from hypertensive rats.Release of C-type natriuretic peptide accounts for the biological activity of endothelium-derived hyperpolarizing factor.Membrane hyperpolarization is not required for sustained muscarinic agonist-induced increases in intracellular Ca2+ in arteriolar endothelial cells Impairment of both nitric oxide-mediated and EDHF-type relaxation in small mesenteric arteries from rats with streptozotocin-induced diabetes.Pre-weaning growth hormone treatment reverses hypertension and endothelial dysfunction in adult male offspring of mothers undernourished during pregnancy.Endothelium-derived hyperpolarizing factor: is there a novel chemical mediator?EDHF: spreading the influence of the endothelium.Myoendothelial electrical coupling in arteries and arterioles and its implications for endothelium-derived hyperpolarizing factor.Role of gap junctions and EETs in endothelium-dependent hyperpolarization of porcine coronary artery The role of gap junctions in mediating endothelium-dependent responses to bradykinin in myometrial small arteries isolated from pregnant women.Endothelium-dependent relaxation and endothelial hyperpolarization by P2Y receptor agonists in rat-isolated mesenteric artery.Ouabain exerts biphasic effects on connexin functionality and expression in vascular smooth muscle cells Reciprocal changes in endothelium-derived hyperpolarizing factor- and nitric oxide-system in the mesenteric artery of adult female rats following ovariectomy.5-hydroxytryptamine has an endothelium-derived hyperpolarizing factor-like effect on coronary flow in isolated rat hearts.Role of endothelium-derived hyperpolarizing factor in endothelial dysfunction during diabetes.C-type natriuretic peptide and endothelium-dependent hyperpolarization in the guinea-pig carotid artery.Role of potassium in regulating blood flow and blood pressure.Endothelium-dependent hyperpolarizations: past beliefs and present facts.Therapeutic manipulation of glucocorticoid metabolism in cardiovascular disease Upregulation of intermediate calcium-activated potassium channels counterbalance the impaired endothelium-dependent vasodilation in stroke-prone spontaneously hypertensive rats.EDHF: an update.Endothelial control of vasodilation: integration of myoendothelial microdomain signalling and modulation by epoxyeicosatrienoic acids.Endothelium-derived Relaxing Factors of Small Resistance Arteries in Hypertension.Communication Is Key: Mechanisms of Intercellular Signaling in Vasodilation.The expression and function of Ca(2+)-sensing receptors in rat mesenteric artery; comparative studies using a model of type II diabetes Smooth muscle Ca(2+) -activated and voltage-gated K+ channels modulate conducted dilation in rat isolated small mesenteric arteries.Gap junctional communication underpins EDHF-type relaxations evoked by ACh in the rat hepatic artery.The extracellular regulated kinases (ERK) 1/2 mediate cannabinoid-induced inhibition of gap junctional communication in endothelial cells.A novel role for HNO in local and spreading vasodilatation in rat mesenteric resistance arteries.Impairment of endothelial SK(Ca) channels and of downstream hyperpolarizing pathways in mesenteric arteries from spontaneously hypertensive rats.

P2860

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P2860

Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.

http://www.wikidata.org/entity/Q42735742

description

1999 nî lūn-bûn

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1999年の論文

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1999年論文

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1999年論文

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1999年論文

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1999年論文

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1999年論文

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1999年论文

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1999年论文

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1999年论文

@zh-cn

name

Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.

@en

Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.

@nl

type

label

Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.

@en

Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.

@nl

prefLabel

Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.

@en

Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.

@nl

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British Journal of Pharmacology

P1476

Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.

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P2093

A H Weston

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C Thollon

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G Edwards

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M Félétou

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M J Gardener

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P M Vanhoutte

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P2860

Elevation of intracellular calcium in smooth muscle causes endothelial cell generation of NO in arterioles.

Potassium ions and endothelium-derived hyperpolarizing factor in guinea-pig carotid and porcine coronary arteries

Endothelium-dependent hyperpolarization: a role in the control of vascular tone.

Differential regulation of Na,K-ATPase isozymes by protein kinases and arachidonic acid.

Regulation of the alpha 2 beta 1 and alpha 3 beta 1 isozymes of the Na,K-ATPase by Ca2+, PKA, and PKC.

Gap junctions in vascular tissues. Evaluating the role of intercellular communication in the modulation of vasomotor tone.

Gap-junction disassembly and connexin 43 dephosphorylation induced by 18 beta-glycyrrhetinic acid.

Endothelium-derived hyperpolarizing factor(s): updating the unknown.

Endothelium-derived factors and hyperpolarization of the carotid artery of the guinea-pig.

The role of myoendothelial cell contact in non-nitric oxide-, non-prostanoid-mediated endothelium-dependent relaxation of porcine coronary artery.

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1788-1794

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10.1038/SJ.BJP.0703009

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8

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128

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12712268

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10588935

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1571823

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