Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.
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Endothelium-dependent smooth muscle hyperpolarization: do gap junctions provide a unifying hypothesis?Inducible endothelium-derived hyperpolarizing factor: role of the 15-lipoxygenase-EDHF pathwayFurther investigations into the endothelium-dependent hyperpolarizing effects of bradykinin and substance P in porcine coronary arteryBlockade of chloride channels reveals relaxations of rat small mesenteric arteries to raised potassiumBradykinin attenuates the [Ca(2+)](i) response to angiotensin II of renal juxtamedullary efferent arterioles via an EDHFSuppression of K(+)-induced hyperpolarization by phenylephrine in rat mesenteric artery: relevance to studies of endothelium-derived hyperpolarizing factorAlterations in EDHF-mediated hyperpolarization and relaxation in mesenteric arteries of female rats in long-term deficiency of oestrogen and during oestrus cycleK+ currents underlying the action of endothelium-derived hyperpolarizing factor in guinea-pig, rat and human blood vesselsRegulation of cellular communication by signaling microdomains in the blood vessel wall.Modulation of endothelial cell KCa3.1 channels during endothelium-derived hyperpolarizing factor signaling in mesenteric resistance arteries.Angiotensin II receptor blockade corrects altered expression of gap junctions in vascular endothelial cells from hypertensive rats.Release of C-type natriuretic peptide accounts for the biological activity of endothelium-derived hyperpolarizing factor.Membrane hyperpolarization is not required for sustained muscarinic agonist-induced increases in intracellular Ca2+ in arteriolar endothelial cellsImpairment of both nitric oxide-mediated and EDHF-type relaxation in small mesenteric arteries from rats with streptozotocin-induced diabetes.Pre-weaning growth hormone treatment reverses hypertension and endothelial dysfunction in adult male offspring of mothers undernourished during pregnancy.Endothelium-derived hyperpolarizing factor: is there a novel chemical mediator?EDHF: spreading the influence of the endothelium.Myoendothelial electrical coupling in arteries and arterioles and its implications for endothelium-derived hyperpolarizing factor.Role of gap junctions and EETs in endothelium-dependent hyperpolarization of porcine coronary arteryThe role of gap junctions in mediating endothelium-dependent responses to bradykinin in myometrial small arteries isolated from pregnant women.Endothelium-dependent relaxation and endothelial hyperpolarization by P2Y receptor agonists in rat-isolated mesenteric artery.Ouabain exerts biphasic effects on connexin functionality and expression in vascular smooth muscle cellsReciprocal changes in endothelium-derived hyperpolarizing factor- and nitric oxide-system in the mesenteric artery of adult female rats following ovariectomy.5-hydroxytryptamine has an endothelium-derived hyperpolarizing factor-like effect on coronary flow in isolated rat hearts.Role of endothelium-derived hyperpolarizing factor in endothelial dysfunction during diabetes.C-type natriuretic peptide and endothelium-dependent hyperpolarization in the guinea-pig carotid artery.Role of potassium in regulating blood flow and blood pressure.Endothelium-dependent hyperpolarizations: past beliefs and present facts.Therapeutic manipulation of glucocorticoid metabolism in cardiovascular diseaseUpregulation of intermediate calcium-activated potassium channels counterbalance the impaired endothelium-dependent vasodilation in stroke-prone spontaneously hypertensive rats.EDHF: an update.Endothelial control of vasodilation: integration of myoendothelial microdomain signalling and modulation by epoxyeicosatrienoic acids.Endothelium-derived Relaxing Factors of Small Resistance Arteries in Hypertension.Communication Is Key: Mechanisms of Intercellular Signaling in Vasodilation.The expression and function of Ca(2+)-sensing receptors in rat mesenteric artery; comparative studies using a model of type II diabetesSmooth muscle Ca(2+) -activated and voltage-gated K+ channels modulate conducted dilation in rat isolated small mesenteric arteries.Gap junctional communication underpins EDHF-type relaxations evoked by ACh in the rat hepatic artery.The extracellular regulated kinases (ERK) 1/2 mediate cannabinoid-induced inhibition of gap junctional communication in endothelial cells.A novel role for HNO in local and spreading vasodilatation in rat mesenteric resistance arteries.Impairment of endothelial SK(Ca) channels and of downstream hyperpolarizing pathways in mesenteric arteries from spontaneously hypertensive rats.
P2860
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P2860
Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.
@en
Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.
@nl
type
label
Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.
@en
Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.
@nl
prefLabel
Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.
@en
Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.
@nl
P2093
P2860
P356
P1476
Role of gap junctions in the responses to EDHF in rat and guinea-pig small arteries.
@en
P2093
A H Weston
M J Gardener
P M Vanhoutte
P2860
P304
P356
10.1038/SJ.BJP.0703009
P407
P577
1999-12-01T00:00:00Z