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Advances in Skeletal Dysplasia GeneticsDual pathways to endochondral osteoblasts: a novel chondrocyte-derived osteoprogenitor cell identified in hypertrophic cartilageDeficiency and Also Transgenic Overexpression of Timp-3 Both Lead to Compromised Bone Mass and Architecture In Vivo.Chondrocytes Directly Transform into Bone Cells in Mandibular Condyle Growth.The Mighty Chondrocyte: No Bones about It.β-catenin activity in late hypertrophic chondrocytes locally orchestrates osteoblastogenesis and osteoclastogenesis.Deformed Skull Morphology Is Caused by the Combined Effects of the Maldevelopment of Calvarias, Cranial Base and Brain in FGFR2-P253R Mice Mimicking Human Apert Syndrome.Fate of growth plate hypertrophic chondrocytes: death or lineage extension?Chondrogenesis and osteogenesis are one continuous developmental and lineage defined biological process.Expression of RMRP RNA is regulated in chondrocyte hypertrophy and determines chondrogenic differentiation.Reverse engineering development: crosstalk opportunities between developmental biology and tissue engineering.microRNA Regulation of Skeletal Development.Inducible activation of FGFR2 in adult mice promotes bone formation after bone marrow ablation.Co-localization of Cell Lineage Markers and the Tomato Signal.BMP signalling in skeletal development, disease and repair.Roles of Chondrocytes in Endochondral Bone Formation and Fracture Repair.Gdf5 progenitors give rise to fibrocartilage cells that mineralize via hedgehog signaling to form the zonal enthesis.An Emerging Regulatory Landscape for Skeletal Development.Cartilage to bone transformation during fracture healing is coordinated by the invading vasculature and induction of the core pluripotency genes.Evolutionary origin of endochondral ossification: the transdifferentiation hypothesis.Achondroplasia: Development, pathogenesis, and therapy.Pannexin 3 is required for late stage bone growth but not for initiation of ossification in avian embryos.FGFR3 deficient mice have accelerated fracture repair.Chondrocyte FGFR3 Regulates Bone Mass by Inhibiting Osteogenesis.Dysregulated PDGFRα signaling alters coronal suture morphogenesis and leads to craniosynostosis through endochondral ossification.Lineage tracing reveals conversion of liver sinusoidal endothelial cells into hepatocytes.Indomethacin induces differential effects on in vitro endochondral ossification depending on the chondrocyte's differentiation stage.Vital Roles of β-catenin in Trans-differentiation of Chondrocytes to Bone Cells.Osteogenic Treatment Initiating a Tissue-Engineered Cartilage Template Hypertrophic Transition.Synergistic co-regulation and competition by a SOX9-GLI-FOXA phasic transcriptional network coordinate chondrocyte differentiation transitions.
P2860
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P2860
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
2014年论文
@zh
2014年论文
@zh-cn
name
Osteogenic fate of hypertrophic chondrocytes.
@en
Osteogenic fate of hypertrophic chondrocytes.
@nl
type
label
Osteogenic fate of hypertrophic chondrocytes.
@en
Osteogenic fate of hypertrophic chondrocytes.
@nl
prefLabel
Osteogenic fate of hypertrophic chondrocytes.
@en
Osteogenic fate of hypertrophic chondrocytes.
@nl
P2093
P2860
P356
P1433
P1476
Osteogenic fate of hypertrophic chondrocytes
@en
P2093
P2860
P2888
P304
P356
10.1038/CR.2014.111
P577
2014-08-22T00:00:00Z
P5875
P6179
1012440006