about
Hypoxia inactivates inducible nitric oxide synthase in mouse macrophages by disrupting its interaction with alpha-actinin 4Dose-related effects of hyperoxia on the lung inflammatory response in septic rats.Loss of inducible nitric oxide synthase expression in the mouse renal cell carcinoma cell line RENCA is mediated by microRNA miR-146a.The use of a single serological marker underestimates the prevalence of celiac disease in Israel: a study of blood donors.An epitope-specific novel anti-EMMPRIN polyclonal antibody inhibits tumor progression.Molecular mechanisms regulating macrophage response to hypoxia.Hypoxia increases membranal and secreted HLA-DR in endothelial cells, rendering them T-cell activators.Reduced TIMP-2 in hypoxia enhances angiogenesis.Native and fragmented fibronectin oppositely modulate monocyte secretion of MMP-9.Hypoxia reduces the output of matrix metalloproteinase-9 (MMP-9) in monocytes by inhibiting its secretion and elevating membranal association.The role of IL-18 and IL-12 in the modulation of matrix metalloproteinases and their tissue inhibitors in monocytic cells.Inorganic lead enhances cytokine-induced elevation of matrix metalloproteinase MMP-9 expression in glial cells.Intravenous gamma globulin inhibits the production of matrix metalloproteinase-9 in macrophages.Regulation of endothelial matrix metalloproteinase-2 by hypoxia/reoxygenation.
P50
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P50
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Nitza Lahat
@ast
Nitza Lahat
@en
Nitza Lahat
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Nitza Lahat
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Nitza Lahat
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Nitza Lahat
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Nitza Lahat
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Nitza Lahat
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Nitza Lahat
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Nitza Lahat
@en
Nitza Lahat
@es
Nitza Lahat
@nl