'(De-)sensitization' vs. 'Uncoupling': what drives cardiomyopathies in the thin filament? Reply.

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The Importance of Intrinsically Disordered Segments of Cardiac Troponin in Modulating Function by Phosphorylation and Disease-Causing Mutations

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'(De-)sensitization' vs. 'Uncoupling': what drives cardiomyopathies in the thin filament? Reply.

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'(De-)sensitization' vs. 'Unco ...... s in the thin filament? Reply.

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'(De-)sensitization' vs. 'Unco ...... s in the thin filament? Reply.

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'(De-)sensitization' vs. 'Unco ...... s in the thin filament? Reply.

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'(De-)sensitization' vs. 'Unco ...... es in the thin filament? Reply

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Andrew Messer

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Maria Papadaki

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Structure of the C-domain of human cardiac troponin C in complex with the Ca2+ sensitizing drug EMD 57033

Phosphorylation-dependent conformational transition of the cardiac specific N-extension of troponin I in cardiac troponin

Solution Structure of Human Cardiac Troponin C in Complex with the Green Tea Polyphenol, (-)-Epigallocatechin 3-Gallate

The cardiac-specific N-terminal region of troponin I positions the regulatory domain of troponin C

Targeting the sarcomere to correct muscle function

Investigating the role of uncoupling of troponin I phosphorylation from changes in myofibrillar Ca(2+)-sensitivity in the pathogenesis of cardiomyopathy.

The dilated cardiomyopathy-causing mutation ACTC E361G in cardiac muscle myofibrils specifically abolishes modulation of Ca(2+) regulation by phosphorylation of troponin I

The unique functions of cardiac troponin I in the control of cardiac muscle contraction and relaxation.

How do mutations in contractile proteins cause the primary familial cardiomyopathies?

Characterization of the interaction between the N-terminal extension of human cardiac troponin I and troponin C.

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187-188

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scholarly article

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10.1093/CVR/CVV233

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109

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Steven B Marston

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2015-10-13T00:00:00Z

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282872559

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26464332

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4888944

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'(De-)sensitization' vs. 'Uncoupling': what drives cardiomyopathies in the thin filament? Reply.

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'(De-)sensitization' vs. 'Uncoupling': what drives cardiomyopathies in the thin filament? Reply.

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