Glycosylphosphatidylinositol-linked ceruloplasmin is expressed in multiple rodent organs and is lower following dietary copper deficiency.
about
Charting the travels of copper in eukaryotes from yeast to mammalsMolecular mediators governing iron-copper interactionsTranscriptional regulation of copper metabolism genes in the liver of fetal and neonatal control and iron-deficient ratsThe role of subcutaneous adipose tissue in supporting the copper balance in rats with a chronic deficiency in holo-ceruloplasmin.Functional properties of human ferroportin, a cellular iron exporter reactive also with cobalt and zinc.The potential for transition metal-mediated neurodegeneration in amyotrophic lateral sclerosisImpact of copper limitation on expression and function of multicopper oxidases (ferroxidases)Mechanism of Copper Uptake from Blood Plasma Ceruloplasmin by Mammalian Cells.Suppressed hepcidin expression correlates with hypotransferrinemia in copper-deficient rat pups but not dams.Copper chelation and interleukin-6 proinflammatory cytokine effects on expression of different proteins involved in iron metabolism in HepG2 cell line.Impairment of interrelated iron- and copper homeostatic mechanisms in brain contributes to the pathogenesis of neurodegenerative disorders.Intragastric exposure to titanium dioxide nanoparticles induced nephrotoxicity in mice, assessed by physiological and gene expression modifications.Hephaestin and ceruloplasmin facilitate iron metabolism in the mouse kidney.Impact of copper deficiency in humans.The Ferroxidase Hephaestin But Not Amyloid Precursor Protein is Required for Ferroportin-Supported Iron Efflux in Primary Hippocampal Neurons.Ceruloplasmin is regulated by copper and lactational hormones in PMC42-LA mammary epithelial cell culture models.Copper deficiency has minimal impact on ferroportin expression or function.Ceruloplasmin and hephaestin jointly protect the exocrine pancreas against oxidative damage by facilitating iron efflux.
P2860
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P2860
Glycosylphosphatidylinositol-linked ceruloplasmin is expressed in multiple rodent organs and is lower following dietary copper deficiency.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Glycosylphosphatidylinositol-l ...... ing dietary copper deficiency.
@en
Glycosylphosphatidylinositol-l ...... ing dietary copper deficiency.
@nl
type
label
Glycosylphosphatidylinositol-l ...... ing dietary copper deficiency.
@en
Glycosylphosphatidylinositol-l ...... ing dietary copper deficiency.
@nl
prefLabel
Glycosylphosphatidylinositol-l ...... ing dietary copper deficiency.
@en
Glycosylphosphatidylinositol-l ...... ing dietary copper deficiency.
@nl
P2860
P356
P1476
Glycosylphosphatidylinositol-l ...... ing dietary copper deficiency.
@en
P2093
Elise J Mostad
Joseph R Prohaska
P2860
P304
P356
10.1258/EBM.2010.010256
P577
2011-02-25T00:00:00Z