Reprogramming aging through DOT1L inhibition - Wikidata - awgldk - TriplyDB

Reprogramming aging through DOT1L inhibition

Reprogramming aging through DOT1L inhibition

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Hallmarks of progeroid syndromes: lessons from mice and reprogrammed cells Senescence-Inflammatory Regulation of Reparative Cellular Reprogramming in Aging and Cancer.

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Reprogramming aging through DOT1L inhibition

http://www.wikidata.org/entity/Q42753695

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

@zh-tw

2015年论文

@wuu

2015年论文

@zh

2015年论文

@zh-cn

name

Reprogramming aging through DOT1L inhibition

@en

Reprogramming aging through DOT1L inhibition

@nl

type

label

Reprogramming aging through DOT1L inhibition

@en

Reprogramming aging through DOT1L inhibition

@nl

prefLabel

Reprogramming aging through DOT1L inhibition

@en

Reprogramming aging through DOT1L inhibition

@nl

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15384101.2015.1093443

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Reprogramming aging through DOT1L inhibition

@en

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Clara Soria-Valles

http://www.w3.org/2001/XMLSchema#string

Fernando G Osorio

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Exome sequencing and functional analysis identifies BANF1 mutation as the cause of a hereditary progeroid syndrome

The hallmarks of aging

Chromatin-modifying enzymes as modulators of reprogramming

Progressive methylation of ageing histones by Dot1 functions as a timer.

NF-κB activation impairs somatic cell reprogramming in ageing.

Nuclear lamina defects cause ATM-dependent NF-κB activation and link accelerated aging to a systemic inflammatory response.

Nuclear envelope alterations generate an aging-like epigenetic pattern in mice deficient in Zmpste24 metalloprotease

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3345-3346

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scholarly article

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10.1080/15384101.2015.1093443

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21

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14

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Carlos López Otín

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2015-09-16T00:00:00Z

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282041074

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26375309

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4825545

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