Detrimental effects of antiapoptotic treatments in spinal cord injury.
about
An anti-apoptotic peptide improves survival in lethal total body irradiation.Targeting Bcl-2 based on the interaction of its BH4 domain with the inositol 1,4,5-trisphosphate receptor.Lipopolysaccharide preconditioning attenuates neuroapoptosis and improves functional recovery through activation of Nrf2 in traumatic spinal cord injury rats.Apoptotic cell death and altered calcium homeostasis caused by frataxin depletion in dorsal root ganglia neurons can be prevented by BH4 domain of Bcl-xL protein.Lipopolysaccharide preconditioning attenuates apoptotic processes and improves neuropathologic changes after spinal cord injury in rats.
P2860
Detrimental effects of antiapoptotic treatments in spinal cord injury.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Detrimental effects of antiapoptotic treatments in spinal cord injury.
@en
Detrimental effects of antiapoptotic treatments in spinal cord injury.
@nl
type
label
Detrimental effects of antiapoptotic treatments in spinal cord injury.
@en
Detrimental effects of antiapoptotic treatments in spinal cord injury.
@nl
prefLabel
Detrimental effects of antiapoptotic treatments in spinal cord injury.
@en
Detrimental effects of antiapoptotic treatments in spinal cord injury.
@nl
P2093
P2860
P1476
Detrimental effects of antiapoptotic treatments in spinal cord injury
@en
P2093
Claire Hulsebosch
J Regino Perez-Polo
Kathia Johnson
Olivera Nesic
P2860
P304
P356
10.1016/J.EXPNEUROL.2007.03.001
P407
P577
2007-03-07T00:00:00Z